Interleukin-1 receptor-induced PGE
Acetylcholine
/ metabolism
Animals
Antivenins
/ administration & dosage
Atropine
/ pharmacology
Dexamethasone
/ administration & dosage
Dinoprostone
/ biosynthesis
Disease Models, Animal
Heart Failure
/ drug therapy
Humans
Inflammation Mediators
/ metabolism
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Models, Cardiovascular
Neuroimmunomodulation
/ drug effects
Receptors, Interleukin-1 Type I
/ deficiency
Scorpion Stings
/ complications
Scorpion Venoms
/ toxicity
Scorpions
Signal Transduction
Vagotomy
Journal
Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555
Informations de publication
Date de publication:
28 10 2020
28 10 2020
Historique:
received:
06
09
2019
accepted:
25
09
2020
entrez:
29
10
2020
pubmed:
30
10
2020
medline:
11
11
2020
Statut:
epublish
Résumé
Scorpion envenomation is a leading cause of morbidity and mortality among accidents caused by venomous animals. Major clinical manifestations that precede death after scorpion envenomation include heart failure and pulmonary edema. Here, we demonstrate that cardiac dysfunction and fatal outcomes caused by lethal scorpion envenomation in mice are mediated by a neuro-immune interaction linking IL-1 receptor signaling, prostaglandin E
Identifiants
pubmed: 33116136
doi: 10.1038/s41467-020-19232-8
pii: 10.1038/s41467-020-19232-8
pmc: PMC7595177
doi:
Substances chimiques
Antivenins
0
IL1R1 protein, mouse
0
Inflammation Mediators
0
Receptors, Interleukin-1 Type I
0
Scorpion Venoms
0
Atropine
7C0697DR9I
Dexamethasone
7S5I7G3JQL
Dinoprostone
K7Q1JQR04M
Acetylcholine
N9YNS0M02X
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
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