Alterations of serum levels of plasminogen, TNF-α, and IDO in granulomatosis with polyangiitis patients.


Journal

Vascular
ISSN: 1708-539X
Titre abrégé: Vascular
Pays: England
ID NLM: 101196722

Informations de publication

Date de publication:
Dec 2021
Historique:
pubmed: 12 1 2021
medline: 25 11 2021
entrez: 11 1 2021
Statut: ppublish

Résumé

Granulomatosis with polyangiitis (GPA) is a representative of vasculitides associated with anti-neutrophil cytoplasmic autoantibodies. "Classical" antibodies directed against proteinase 3 are involved in the pathogenesis and are part of the GPA diagnosis at the same time. Along with them, however, antibodies against Lysosomal-Associated Membrane Protein-2 (LAMP-2) and antibodies directed against plasminogen have been described in GPA. The results showed that in GPA, serum plasminogen levels were negatively associated with renal involvement (receiver operating characteristic (ROC) area under the curve (AUC) of 0.78) (95% CI 0.53-0.91), In this study, we demonstrate the alteration of soluble factors, which play an important role in the pathogenesis of GPA and their relationship with the clinical manifestations of the disease. Our main results confirm the associations of increased secretory TNF-α and some clinical manifestations, and we describe for the first time decreased serum plasminogen levels and their association with renal involvement.

Sections du résumé

BACKGROUND BACKGROUND
Granulomatosis with polyangiitis (GPA) is a representative of vasculitides associated with anti-neutrophil cytoplasmic autoantibodies. "Classical" antibodies directed against proteinase 3 are involved in the pathogenesis and are part of the GPA diagnosis at the same time. Along with them, however, antibodies against Lysosomal-Associated Membrane Protein-2 (LAMP-2) and antibodies directed against plasminogen have been described in GPA.
RESULTS RESULTS
The results showed that in GPA, serum plasminogen levels were negatively associated with renal involvement (receiver operating characteristic (ROC) area under the curve (AUC) of 0.78) (95% CI 0.53-0.91),
CONCLUSIONS CONCLUSIONS
In this study, we demonstrate the alteration of soluble factors, which play an important role in the pathogenesis of GPA and their relationship with the clinical manifestations of the disease. Our main results confirm the associations of increased secretory TNF-α and some clinical manifestations, and we describe for the first time decreased serum plasminogen levels and their association with renal involvement.

Identifiants

pubmed: 33427113
doi: 10.1177/1708538120986305
doi:

Substances chimiques

Biomarkers 0
Indoleamine-Pyrrole 2,3,-Dioxygenase 0
LAMP2 protein, human 0
Lysosomal-Associated Membrane Protein 2 0
TNF protein, human 0
Tumor Necrosis Factor-alpha 0
Plasminogen 9001-91-6

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

874-882

Auteurs

Dobroslav Kyurkchiev (D)

Laboratory of Clinical immunology, University Hospital St. Ivan Rilski, Department of Clinical Immunology, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Tsvetelina Yoneva (T)

Department of Rheumatology, Clinic of Rheumatology, University Hospital St. Ivan Rilski, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Adelina Yordanova (A)

Laboratory of Clinical immunology, University Hospital St. Ivan Rilski, Department of Clinical Immunology, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Ekaterina Kurteva (E)

Laboratory of Clinical immunology, University Hospital St. Ivan Rilski, Department of Clinical Immunology, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Georgi Vasilev (G)

Laboratory of Clinical immunology, University Hospital St. Ivan Rilski, Department of Clinical Immunology, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Yana Zdravkova (Y)

Department of Rheumatology, Clinic of Rheumatology, University Hospital St. Ivan Rilski, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Ivan Sheytanov (I)

Department of Rheumatology, Clinic of Rheumatology, University Hospital St. Ivan Rilski, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Rasho Rashkov (R)

Department of Rheumatology, Clinic of Rheumatology, University Hospital St. Ivan Rilski, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

Ekaterina Ivanova-Todorova (E)

Laboratory of Clinical immunology, University Hospital St. Ivan Rilski, Department of Clinical Immunology, Medical Faculty, Medical University of Sofia, Sofia, Bulgaria.

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Classifications MeSH