FATC Domain Deletion Compromises ATM Protein Stability, Blocks Lymphocyte Development, and Promotes Lymphomagenesis.


Journal

Journal of immunology (Baltimore, Md. : 1950)
ISSN: 1550-6606
Titre abrégé: J Immunol
Pays: United States
ID NLM: 2985117R

Informations de publication

Date de publication:
15 03 2021
Historique:
received: 20 08 2020
accepted: 04 01 2021
pubmed: 5 2 2021
medline: 6 8 2021
entrez: 4 2 2021
Statut: ppublish

Résumé

Ataxia-telangiectasia mutated (ATM) kinase is a master regulator of the DNA damage response, and loss of ATM leads to primary immunodeficiency and greatly increased risk for lymphoid malignancies. The FATC domain is conserved in phosphatidylinositol-3-kinase-related protein kinases (PIKKs). Truncation mutation in the FATC domain (R3047X) selectively compromised reactive oxygen species-induced ATM activation in cell-free assays. In this article, we show that in mouse models, knock-in ATM-R3057X mutation (

Identifiants

pubmed: 33536256
pii: jimmunol.2000967
doi: 10.4049/jimmunol.2000967
pmc: PMC9305077
mid: NIHMS1661647
doi:

Substances chimiques

Codon, Nonsense 0
Ataxia Telangiectasia Mutated Proteins EC 2.7.11.1
Atm protein, mouse EC 2.7.11.1

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1228-1239

Subventions

Organisme : NCI NIH HHS
ID : R01 CA226852
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA013696
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM102362
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA158073
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA184187
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA215067
Pays : United States
Organisme : NCI NIH HHS
ID : P01 CA174653
Pays : United States

Informations de copyright

Copyright © 2021 by The American Association of Immunologists, Inc.

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Auteurs

Maja Milanovic (M)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Zhengping Shao (Z)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Verna M Estes (VM)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Xiaobin S Wang (XS)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.
Department of Pathology and Cell Biology, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Demis Menolfi (D)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Xiaohui Lin (X)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Brian J Lee (BJ)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Jun Xu (J)

Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California San Diego, La Jolla, CA 92093.

Olivia M Cupo (OM)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

Dong Wang (D)

Skaggs School of Pharmacy and Pharmaceutical Sciences, University of California San Diego, La Jolla, CA 92093.

Shan Zha (S)

Institute for Cancer Genetics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032; sz2296@cumc.columbia.edu.
Department of Pathology and Cell Biology, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.
Division of Pediatric Oncology, Hematology and Stem Cell Transplantation, Department of Pediatrics, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032; and.
Department of Immunology and Microbiology, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY 10032.

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