Life-Threatening Influenza, Hemophagocytic Lymphohistiocytosis and Probable Vaccine-Strain Varicella in a Novel Case of Homozygous

JAK-STAT signaling pathway hyperinflammation inborn errors of antiviral immunity interferon stimulated gene type I and type III interferon signaling type I interferon varicella zoster virus viral disease

Journal

Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960

Informations de publication

Date de publication:
2020
Historique:
received: 31 10 2020
accepted: 29 12 2020
entrez: 8 3 2021
pubmed: 9 3 2021
medline: 25 6 2021
Statut: epublish

Résumé

STAT2 is a transcription factor that plays an essential role in antiviral immunity by mediating the activity of type I and III interferons (IFN-I and IFN-III). It also has a recently established function in the negative regulation of IFN-I signaling. Homozygous STAT2 deficiency is an ultra-rare inborn error of immunity which provides unique insight into the pathologic consequence of STAT2 dysfunction. We report here a novel genetic cause of homozygous STAT2 deficiency with several notable clinical features. The proband presented aged 12 months with hemophagocytic lymphohistiocytosis (HLH) closely followed by clinical varicella, both occurring within three weeks of measles, mumps, and rubella (MMR) and varicella vaccinations. There was a history of life-threatening influenza A virus (IAV) disease 2 months previously. Genetic investigation uncovered homozygosity for a novel nonsense variant in

Identifiants

pubmed: 33679716
doi: 10.3389/fimmu.2020.624415
pmc: PMC7930908
doi:

Substances chimiques

Chickenpox Vaccine 0
Codon, Nonsense 0
STAT2 Transcription Factor 0

Types de publication

Case Reports Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

624415

Subventions

Organisme : Wellcome Trust
ID : 207556/Z/17/Z
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 211153/Z/18/Z
Pays : United Kingdom

Informations de copyright

Copyright © 2021 Freij, Hanrath, Chen, Hambleton and Duncan.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Bishara J Freij (BJ)

Pediatric Department, Beaumont Children's Hospital, Royal Oak, MI, United States.
Oakland University William Beaumont School of Medicine, Rochester, MI, United States.

Aidan T Hanrath (AT)

Immunity and Inflammation Theme, Translational and Clinical Research Institute, Newcastle upon Tyne, United Kingdom.

Rui Chen (R)

Immunity and Inflammation Theme, Translational and Clinical Research Institute, Newcastle upon Tyne, United Kingdom.

Sophie Hambleton (S)

Immunity and Inflammation Theme, Translational and Clinical Research Institute, Newcastle upon Tyne, United Kingdom.
Great North Children's Hospital, Newcastle upon Tyne Hospitals NHS Foundation Trust, Newcastle upon Tyne, United Kingdom.

Christopher J A Duncan (CJA)

Immunity and Inflammation Theme, Translational and Clinical Research Institute, Newcastle upon Tyne, United Kingdom.
Royal Victoria Infirmary, Newcastle upon Tyne Hospitals NHS Foundation Trust, Newcastle upon Tyne, United Kingdom.

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Classifications MeSH