COVID-19 in Patients with Multiple Sclerosis: Associations with Disease-Modifying Therapies.


Journal

CNS drugs
ISSN: 1179-1934
Titre abrégé: CNS Drugs
Pays: New Zealand
ID NLM: 9431220

Informations de publication

Date de publication:
03 2021
Historique:
accepted: 10 03 2021
pubmed: 21 3 2021
medline: 13 4 2021
entrez: 20 3 2021
Statut: ppublish

Résumé

Disease-modifying therapies (DMTs) for multiple sclerosis (MS) target immunity and have the potential to increase the risk of severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infection and alter its clinical course. We assessed these risks in patients with MS (PwMS). The objective of this study was to describe the overall risk of coronavirus disease 2019 (COVID-19) infection, severe disease course, and potential population-level predictors of COVID-19 infection in PwMS, and to provide a context using a cohort of patients with systemic lupus erythematosus (SLE). In addition, the association of different MS DMTs with the incidence and clinical course of COVID-19 was evaluated. Safety data from the Biogen Global Safety Database are also presented on reported cases of COVID-19 in patients treated with Biogen MS therapies. The IBM 30,478 PwMS with an open DMT prescription were identified within Explorys; 344 were COVID-19 positive. The most significant risk factors for acquiring COVID-19 were comorbidity score ≥ 1, body mass index ≥ 30, and Black/African ancestry. Similar risk factors were also identified for patients with SLE. Patients with MS were less likely to develop COVID-19 when treated with interferons (0.61%) and glatiramer acetate (0.51%), vs all other MS DMTs (both p < 0.001); anti-CD20 therapy was associated with the highest risk (3.45%; p < 0.0001). In the Biogen Global Safety Database, we identified 1217 patients who were COVID-19 positive treated with intramuscular interferon beta-1a, peginterferon beta-1a, natalizumab, dimethyl fumarate, diroximel fumarate, or fampridine. Comorbidities, obesity, and Black/African ancestry, but not age, were associated with a higher risk of SARS-CoV-2 infection in PwMS. Interferons and glatiramer acetate were associated with a reduced COVID-19 risk, whereas anti-CD20 therapies were associated with an increased risk, within the treated MS cohort. COVID-19 safety reports for patients receiving Biogen MS therapies were consistent with the Explorys database and MS literature, illustrating the replicability and power of this approach.

Sections du résumé

BACKGROUND
Disease-modifying therapies (DMTs) for multiple sclerosis (MS) target immunity and have the potential to increase the risk of severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infection and alter its clinical course. We assessed these risks in patients with MS (PwMS).
OBJECTIVE
The objective of this study was to describe the overall risk of coronavirus disease 2019 (COVID-19) infection, severe disease course, and potential population-level predictors of COVID-19 infection in PwMS, and to provide a context using a cohort of patients with systemic lupus erythematosus (SLE). In addition, the association of different MS DMTs with the incidence and clinical course of COVID-19 was evaluated. Safety data from the Biogen Global Safety Database are also presented on reported cases of COVID-19 in patients treated with Biogen MS therapies.
METHODS
The IBM
RESULTS
30,478 PwMS with an open DMT prescription were identified within Explorys; 344 were COVID-19 positive. The most significant risk factors for acquiring COVID-19 were comorbidity score ≥ 1, body mass index ≥ 30, and Black/African ancestry. Similar risk factors were also identified for patients with SLE. Patients with MS were less likely to develop COVID-19 when treated with interferons (0.61%) and glatiramer acetate (0.51%), vs all other MS DMTs (both p < 0.001); anti-CD20 therapy was associated with the highest risk (3.45%; p < 0.0001). In the Biogen Global Safety Database, we identified 1217 patients who were COVID-19 positive treated with intramuscular interferon beta-1a, peginterferon beta-1a, natalizumab, dimethyl fumarate, diroximel fumarate, or fampridine.
CONCLUSIONS
Comorbidities, obesity, and Black/African ancestry, but not age, were associated with a higher risk of SARS-CoV-2 infection in PwMS. Interferons and glatiramer acetate were associated with a reduced COVID-19 risk, whereas anti-CD20 therapies were associated with an increased risk, within the treated MS cohort. COVID-19 safety reports for patients receiving Biogen MS therapies were consistent with the Explorys database and MS literature, illustrating the replicability and power of this approach.

Identifiants

pubmed: 33743151
doi: 10.1007/s40263-021-00804-1
pii: 10.1007/s40263-021-00804-1
pmc: PMC7980129
doi:

Substances chimiques

Crotonates 0
Hydroxybutyrates 0
Immunologic Factors 0
Immunosuppressive Agents 0
Natalizumab 0
Nitriles 0
Toluidines 0
teriflunomide 1C058IKG3B
Alemtuzumab 3A189DH42V
Cladribine 47M74X9YT5
Rituximab 4F4X42SYQ6
Interferon-beta 77238-31-4
Cyclosporine 83HN0GTJ6D
Cyclophosphamide 8N3DW7272P
Mitoxantrone BZ114NVM5P
Dimethyl Fumarate FO2303MNI2
Fingolimod Hydrochloride G926EC510T
Mycophenolic Acid HU9DX48N0T
Azathioprine MRK240IY2L
Methotrexate YL5FZ2Y5U1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

317-330

Références

World Health Organization. Pneumonia of unknown cause: China. 2020. https://www.who.int/csr/don/05-january-2020-pneumonia-of-unkown-cause-china/en/ . Accessed 7 Sept 2020.
Fauci AS, Lane HC, Redfield RR. COVID-19: navigating the uncharted. N Engl J Med. 2020;382(13):1268–9.
doi: 10.1056/NEJMe2002387
Ge H, Wang X, Yuan X, Xiao G, Wang C, Deng T, et al. The epidemiology and clinical information about COVID-19. Eur J Clin Microbiol Infect Dis. 2020;39(6):1011–9.
doi: 10.1007/s10096-020-03874-z
Lu R, Zhao X, Li J, Niu P, Yang B, Wu H, et al. Genomic characterisation and epidemiology of 2019 novel coronavirus: implications for virus origins and receptor binding. Lancet. 2020;395(10224):565–74.
doi: 10.1016/S0140-6736(20)30251-8
World Health Organization. WHO coronavirus disease (COVID-19) dashboard. 2020. https://covid19.who.int/?gclid=EAIaIQobChMIz-i4-f2i7AIVjIFaBR0ZXAxOEAAYASAAEgIiDPD_BwE . Accessed 7 Oct 2020.
Wei J, Zhao J, Han M, Meng F, Zhou J. SARS-CoV-2 infection in immunocompromised patients: humoral versus cell-mediated immunity. J Immunother Cancer. 2020;8(2):e000862.
doi: 10.1136/jitc-2020-000862
Ciotti JR, Grebenciucova E, Moss BP, Newsome SD. Multiple sclerosis disease-modifying therapies in the COVID-19 era. Ann Neurol. 2020;88(6):1062–4. https://doi.org/10.1002/ana.25907 .
doi: 10.1002/ana.25907 pubmed: 32951235
Gianfrancesco M, Hyrich KL, Al-Adely S, Carmona L, Danila MI, Gossec L, et al. COVID-19 global rheumatology alliance. Characteristics associated with hospitalisation for COVID-19 in people with rheumatic disease: data from the COVID-19 Global Rheumatology Alliance physician-reported registry. Ann Rheum Dis. 2020;79(7):859–66.
Castelo-Branco A, Chiesa F, Conte S, Bengtsson C, Lee S, Minton N, et al. Infections in patients with multiple sclerosis: a national cohort study in Sweden. Mult Scler Relat Disord. 2020;45:102420.
doi: 10.1016/j.msard.2020.102420
Danza A, Ruiz-Irastorza G. Infection risk in systemic lupus erythematosus patients: susceptibility factors and preventive strategies. Lupus. 2013;22(12):1286–94.
doi: 10.1177/0961203313493032
Boziki MK, Mentis AFA, Shumilina M, Makshakov G, Evdoshenko E, Grigoriadis N. COVID-19 immunopathology and the central nervous system: implication for multiple sclerosis and other autoimmune diseases with associated demyelination. Brain Sci. 2020;10(6):345.
doi: 10.3390/brainsci10060345
MS International Federation. Global COVID-19 advice for people with MS. 2020. http://www.msif.org/wp-content/uploads/2020/06/MSIF-Global-advice-on-COVID-19-for-people-with-MS-_-updated17June2020.pdf . Accessed 21 Oct 2020.
Bhatia R, Srivastava MVP, Khurana D, Pandit L, Mathew T, Gupta S, et al. Consensus statement on immune modulation in multiple sclerosis and related disorders during the COVID-19 pandemic: expert group on behalf of the Indian Academy of Neurology. Ann Indian Acad Neurol. 2020;23(Suppl. 1):S5-14.
pubmed: 32419748 pmcid: 7213028
Centers for Disease Control and Prevention. If you are immunocompromised, protect yourself from COVID-19. 2020. http://www.cdc.gov/coronavirus/2019-ncov/need-extra-precautions/immunocompromised.html . Accessed 21 Oct 2020.
Rae-Grant A, Day GS, Marrie RA, Rabinstein A, Cree BAC, Gronseth GS, et al. Practice guideline recommendations summary: disease-modifying therapies for adults with multiple sclerosis: report of the Guideline Development, Dissemination, and Implementation Subcommittee of the American Academy of Neurology. Neurology. 2018;90(17):777–88.
doi: 10.1212/WNL.0000000000005347
Berger JR, Brandstadter R, Bar-Or A. COVID-19 and MS disease-modifying therapies. Neurol Neuroimmunol Neuroinflamm. 2020;7(4):e761.
doi: 10.1212/NXI.0000000000000761
Fan M, Qiu W, Bu B, Xu Y, Yang H, Huang D, et al. Risk of COVID-19 infection in MS and neuromyelitis optica spectrum disorders. Neurol Neuroimmunol Neuroinflamm. 2020;7(5):e787.
doi: 10.1212/NXI.0000000000000787
Parrotta E, Kister I, Charvet L, Sammarco C, Saha V, Charlson RE, et al. COVID-19 outcomes in MS: observational study of early experience from NYU Multiple Sclerosis Comprehensive Care Center. Neurol Neuroimmunol Neuroinflamm. 2020;7(5):e835.
doi: 10.1212/NXI.0000000000000835
Fernandez-Ruiz R, Masson M, Kim MY, Myers B, Haberman RH, Castillo R, et al. NYU WARCOV Investigators. Leveraging the United States epicenter to provide insights on COVID-19 in patients with systemic lupus erythematosus. Arthritis Rheumatol. 2020;72(12):1971–80.
Zen M, Fuzzi E, Astorri D, Saccon F, Padoan R, Ienna L, et al. SARS-CoV-2 infection in patients with autoimmune rheumatic diseases in northeast Italy: a cross-sectional study on 916 patients. J Autoimmun. 2020;112:102502.
doi: 10.1016/j.jaut.2020.102502
Celius EG. Infections in patients with multiple sclerosis: implications for disease-modifying therapy. Acta Neurol Scand. 2017;136(Suppl. 201):34–6.
doi: 10.1111/ane.12835
Sormani MP. Italian Study Group on COVID-19 infection in multiple sclerosis. An Italian programme for COVID-19 infection in multiple sclerosis. Lancet Neurol. 2020;19(6):481–2.
Giovannoni G, Hawkes C, Lechner-Scott J, Levy M, Waubant E, Gold J. The COVID-19 pandemic and the use of MS disease-modifying therapies. Mult Scler Relat Disord. 2020;39:102073.
doi: 10.1016/j.msard.2020.102073
Sallard E, Lescure FX, Yazdanpanah Y, Mentre F, Peiffer-Smadja N. Type 1 interferons as a potential treatment against COVID-19. Antivir Res. 2020;178:104791.
doi: 10.1016/j.antiviral.2020.104791
Ciotti JR, Valtcheva MV, Cross AH. Effects of MS disease-modifying therapies on responses to vaccinations: a review. Mult Scler Relat Disord. 2020;45:102439.
doi: 10.1016/j.msard.2020.102439
Louapre C, Collongues N, Stankoff B, Giannesini C, Papeix C, Bensa C, et al. Covisep Investigators. Clinical characteristics and outcomes in patients with coronavirus disease 2019 and multiple sclerosis. JAMA Neurol. 2020;77(9):1–10.
Tirupathi R, Muradova V, Shekhar R, Salim SA, Al-Tawfiq JA, Palabindala V. COVID-19 disparity among racial and ethnic minorities in the US: a cross sectional analysis. Travel Med Infect Dis. 2020;38:101904.
doi: 10.1016/j.tmaid.2020.101904
Bassett MT, Chen JT, Krieger N. Variation in racial/ethnic disparities in COVID-19 mortality by age in the United States: a cross-sectional study. PLoS Med. 2020;17(10):e1003402.
doi: 10.1371/journal.pmed.1003402
Zabalza A, Tagliani P, Cárdenas-Robledo S, Arrambide G, Otero-Romero S, Carbonell-Mirabent P, et al. COVID-19 in MS patients: susceptibility and severity risk factors. MSVirtual2020; ECTRIMS/ACTRIMS; 11–13 Sept 2020.
Klineova S, Harel A, Straus Farber R, Zhang Y, Deangelis T, Leung TM, et al. COVID-19 infection in patients with multiple sclerosis: an observational study by The New York COVID-19 Neuro-Immunology Consortium (NYCNIC). MSVirtual2020; ECTRIMS/ACTRIMS; 11-13 Sep 2020.
Loonstra FC, Hoitsma E, van Kempen ZL, Killestein J, Mostert JP. COVID-19 in multiple sclerosis: the Dutch experience. Mult Scler. 2020;26(10):1256–60.
doi: 10.1177/1352458520942198
Chaudhry F, Bulka H, Rathnam AS, Said OM, Lin J, Lorigan H, et al. COVID-19 in multiple sclerosis patients and risk factors for severe infection. J Neurol Sci. 2020;418:117147.
doi: 10.1016/j.jns.2020.117147
Sormani MP, De Rossi N, Schiavetti I, Carmisciano L, Cordioli C, Moiola L, et al. Musc-19 Study Group. Disease modifying therapies and COVID-19 severity in multiple sclerosis. Ann Neurol. 2021. https://doi.org/10.1002/ana.26028 .
IBM. Explorys EHR solutions. https://www.ibm.com/products/explorys-ehr-data-analysis-tools . Accessed 9 Dec 2020.
Mathian A, Mahevas M, Rohmer J, Roumier M, Cohen-Aubart F, Amador-Borrero B, et al. Clinical course of coronavirus disease 2019 (COVID-19) in a series of 17 patients with systemic lupus erythematosus under long-term treatment with hydroxychloroquine. Ann Rheum Dis. 2020;79(6):837–9.
doi: 10.1136/annrheumdis-2020-217566
Najafi S, Rajaei E, Moallemian R, Nokhostin F. The potential similarities of COVID-19 and autoimmune disease pathogenesis and therapeutic options: new insights approach. Clin Rheumatol. 2020;39(11):3223–35.
doi: 10.1007/s10067-020-05376-x
International Conference on Harmonisation of technical requirements for registration of pharmaceuticals for human use. ICH harmonized tripartite guideline: guideline for good clinical practice. J Postgrad Med. 2001;47(1):45–50.
Simpson-Yap S, De Brouwer E, Kalincik T, Rijke N, Hillert J, Walton C, et al. First results of the COVID-19 in MS Global Data Sharing Initiative suggest anti-CD20 DMTs are associated with worse COVID-19 outcomes. MSVirtual2020; ECTRIMS/ACTRIMS; 11–13 Sept 2020.
Mendes MF, Ferreira MI, Sousa NA, Thomaz R, Apóstolos-Pereira SL, Alves-Leon S, et al. Incidence and clinical outcome of COVID-19 in a cohort of 11.560 Brazilian patients with multiple sclerosis. MSVirtual2020; ECTRIMS/ACTRIMS; 11–13 Sept 2020.
Winkelmann A, Loebermann M, Reisinger EC, Hartung HP, Zettl UK. Disease-modifying therapies and infectious risks in multiple sclerosis. Nat Rev Neurol. 2016;12(4):217–33.
doi: 10.1038/nrneurol.2016.21
Zheng C, Kar I, Chen CK, Sau C, Woodson S, Serra A, et al. Multiple sclerosis disease-modifying therapy and the COVID-19 pandemic: implications on the risk of infection and future vaccination. CNS Drugs. 2020;34(9):879–96.
doi: 10.1007/s40263-020-00756-y
Safavi F, Nourbakhsh B, Azimi AR. B-cell depleting therapies may affect susceptibility to acute respiratory illness among patients with multiple sclerosis during the early COVID-19 epidemic in Iran. Mult Scler Relat Disord. 2020;43:102195.
doi: 10.1016/j.msard.2020.102195
Simpson-Yap S, De Brouwer E, Kalincik T, Rijke N, Hillert J, Walton C, et al. Associations of DMT therapies with COVID-19 severity in multiple sclerosis. medRxiv. 2021. https://doi.org/10.1101/2021.02.08.21251316 .
doi: 10.1101/2021.02.08.21251316
Fox RJ, Chan A, Gold R, Phillips JT, Selmaj K, Chang I, et al. Characterizing absolute lymphocyte count profiles in dimethyl fumarate-treated patients with MS: patient management considerations. Neurol Clin Pract. 2016;6(3):220–9.
doi: 10.1212/CPJ.0000000000000238
Naismith RT, Wolinsky JS, Wundes A, LaGanke C, Arnold DL, Obradovic D, et al. Diroximel fumarate (DRF) in patients with relapsing-remitting multiple sclerosis: interim safety and efficacy results from the phase 3 EVOLVE-MS-1 study. Mult Scler. 2020;26(13):1729–39.
doi: 10.1177/1352458519881761
Longbrake EE, Mao-Draayer Y, Cascione M, Zielinski T, Bame E, Brassat D, et al. Dimethyl fumarate treatment shifts the immune environment toward an anti-inflammatory cell profile while maintaining protective humoral immunity. Mult Scler. 2020:1352458520937282.
von Hehn C, Howard J, Liu S, Meka V, Pultz J, Mehta D, et al. Immune response to vaccines is maintained in patients treated with dimethyl fumarate. Neurol Neuroimmunol Neuroinflamm. 2018;5(1):e409.
doi: 10.1212/NXI.0000000000000409
Mehta D, Miller C, Arnold DL, Bame E, Bar-Or A, Gold R, et al. Effect of dimethyl fumarate on lymphocytes in RRMS: Implications for clinical practice. Neurology. 2019;92(15):e1724–38.
doi: 10.1212/WNL.0000000000007262
Schweitzer F, Laurent S, Fink GR, Barnett MH, Hartung HP, Warnke C. Effects of disease-modifying therapy on peripheral leukocytes in patients with multiple sclerosis. J Neurol. 2020. https://doi.org/10.1007/s00415-019-09690-6 .
doi: 10.1007/s00415-019-09690-6 pubmed: 32691236 pmcid: 7370630
Tallantyre EC, Whittam DH, Jolles S, Paling D, Constantinesecu C, Robertson NP, et al. Secondary antibody deficiency: a complication of anti-CD20 therapy for neuroinflammation. J Neurol. 2018;265(5):1115–22.
doi: 10.1007/s00415-018-8812-0
Reder A, Adamo A, Wicklein E-M, Bhatti A. Use and safety of interferon beta-1b during the COVID-19 outbreak: current data from a pharmacovigilance safety database. MSVirtual2020; ECTRIMS/ACTRIMS; 11–13 Sept 2020.
Zhang Q, Bastard P, Liu Z, Le Pen J, Moncada-Velez M, Chen J, et al. Inborn errors of type I IFN immunity in patients with life-threatening COVID-19. Science. 2020;370(6515):eabd4570.
Pairo-Castineira E, Clohisey S, Klaric L, Bretherick A, Rawlik K, Parkinson N, et al. Genetic mechanisms of critical illness in COVID-19. MedRxiv. 2020. https://doi.org/10.1101/2020.09.24.20200048 .
Bastard P, Rosen LB, Zhang Q, Michailidis E, Hoffmann HH, Zhang Y, et al. Auto-antibodies against type I IFNs in patients with life-threatening COVID-19. Science. 2020;370(6515):eabd4585.
Prod’homme T, Zamvil SS. The evolving mechanisms of action of glatiramer acetate. Cold Spring Harb Perspect Med. 2019;9(2):a029249.
doi: 10.1101/cshperspect.a029249
Melnikov M, Sharanova S, Sviridova A, Rogovskii V, Murugina N, Nikolaeva A, et al. The influence of glatiramer acetate on Th17-immune response in multiple sclerosis. PLoS ONE. 2020;15(10):e0240305.
doi: 10.1371/journal.pone.0240305
Hausler D, Hajiyeva Z, Traub JW, Zamvil SS, Lalive PH, Bruck W, et al. Glatiramer acetate immune modulates B-cell antigen presentation in treatment of MS. Neurol Neuroimmunol Neuroinflamm. 2020;7(3):e698.
doi: 10.1212/NXI.0000000000000698
Rommer PS, Milo R, Han MH, Satyanarayan S, Sellner J, Hauer L, et al. Immunological aspects of approved MS therapeutics. Front Immunol. 2019;10:1564.
doi: 10.3389/fimmu.2019.01564
Bar-Or A, Calkwood JC, Chognot C, Evershed J, Fox EJ, Herman A, et al. Effect of ocrelizumab on vaccine responses in patients with multiple sclerosis: the VELOCE study. Neurology. 2020;95(14):e1999-2008.
doi: 10.1212/WNL.0000000000010380
Pescovitz MD, Torgerson TR, Ochs HD, Ocheltree E, McGee P, Krause-Steinrauf H, et al. Effect of rituximab on human in vivo antibody immune responses. J Allergy Clin Immunol. 2011;128(6):1295-302.e5.
doi: 10.1016/j.jaci.2011.08.008
Genentech. Ocrevus highlights of prescribing information. 2020. https://www.gene.com/download/pdf/ocrevus_prescribing.pdf . Accessed 19 Jan 2021.
Mateen FJ, Rezaei S, Alakel N, Gazdag B, Kumar AR, Vogel A. Impact of COVID-19 on U.S. and Canadian neurologists' therapeutic approach to multiple sclerosis: a survey of knowledge, attitudes, and practices. J Neurol. 2020;267(12):3467–75.
National Multiple Sclerosis Society. MS treatment guidelines during the coronavirus pandemic. https://www.nationalmssociety.org/coronavirus-covid-19-information/multiple-sclerosis-and-coronavirus/ms-treatment-guidelines-during-coronavirus . Accessed 8 Feb 2021.
MS-UK. MS and COVID-19. 2021. https://www.ms-uk.org/ms-and-coronavirus . Accessed 25 Jan 2021.
Hughes R, Pedotti R, Koendgen H. COVID-19 in persons with multiple sclerosis treated with ocrelizumab – a pharmacovigilance case series. Mult Scler Relat Disord. 2020;42:102192.
doi: 10.1016/j.msard.2020.102192
Sadeghmousavi S, Rezaei N. COVID-19 and multiple sclerosis: predisposition and precautions in treatment. SN Compr Clin Med. 2020. https://doi.org/10.1007/s42399-020-00504-9 .
doi: 10.1007/s42399-020-00504-9 pubmed: 32895640 pmcid: 7467844

Auteurs

Anthony T Reder (AT)

Department of Neurology and Brain Research Institute, University of Chicago, Chicago, IL, USA. areder@neurology.bsd.uchicago.edu.

Diego Centonze (D)

Laboratory of Synaptic Immunopathology, Department of Systems Medicine, Tor Vergata University, Rome, Italy.
Unit of Neurology, IRCCS Neuromed, Pozzilli, Italy.

Maria L Naylor (ML)

Biogen, Cambridge, MA, USA.

Rajani Rajbhandari (R)

Biogen, Cambridge, MA, USA.

Arman Altincatal (A)

Biogen, Cambridge, MA, USA.

Michelle Kim (M)

Biogen, Cambridge, MA, USA.

Aaron Berdofe (A)

Biogen, Cambridge, MA, USA.

Maha Radhakrishnan (M)

Biogen, Cambridge, MA, USA.

Eunice Jung (E)

Biogen, Cambridge, MA, USA.

Alfred W Sandrock (AW)

Biogen, Cambridge, MA, USA.

Karen Smirnakis (K)

Biogen, Cambridge, MA, USA.

Catrinel Popescu (C)

Biogen Idec, Maidenhead, UK.

Carl de Moor (C)

Biogen, Cambridge, MA, USA.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH