Peanuts or an Isocaloric Lower Fat, Higher Carbohydrate Nighttime Snack Have Similar Effects on Fasting Glucose in Adults with Elevated Fasting Glucose Concentrations: a 6-Week Randomized Crossover Trial.

elevated fasting glucose nutritional intervention peanuts prediabetes randomized controlled trial

Journal

The Journal of nutrition
ISSN: 1541-6100
Titre abrégé: J Nutr
Pays: United States
ID NLM: 0404243

Informations de publication

Date de publication:
11 01 2022
Historique:
received: 26 01 2021
revised: 08 09 2021
accepted: 23 09 2021
pubmed: 26 9 2021
medline: 22 2 2022
entrez: 25 9 2021
Statut: ppublish

Résumé

The glycemic effects of peanuts are not well studied and no trials have been conducted in adults with elevated fasting plasma glucose (FPG). Furthermore, intake of peanuts as a nighttime snack, an eating occasion affecting FPG, has not been examined. The aim was to determine the effect of consuming 28 g/d of peanuts as a nighttime snack for 6 wk on glycemic control and cardiovascular disease risk factors, compared with an isocaloric lower fat, higher carbohydrate (LFHC) snack (whole grain crackers and low-fat cheese), in adults with elevated FPG. In a randomized crossover trial, 50 adults (FPG 100 ± 8 mg/dL) consumed dry roasted, unsalted peanuts [164 kcal; 11% energy (E) carbohydrate, 17% E protein, and 73% E fat] or a LFHC snack (164 kcal; 54% E carbohydrate, 17% E protein, and 33% E fat) in the evening (after dinner and before bedtime) for 6 wk with a 4-wk washout period. Primary (FPG) and secondary end points [Healthy Eating Index-2015 (HEI-2015), weight, insulin, fructosamine, lipids/lipoproteins, central and peripheral blood pressure, and pulse wave velocity] were evaluated at the beginning and end of each condition. Linear mixed models were used for data analysis. FPG was not different between the peanut and LFHC conditions (end point mean difference: -0.6 mg/dL; 95% CI: -2.7, 1.6; P = 0.67). There were no between-condition effects for secondary cardiometabolic endpoints. The HEI-2015 score was not different between the conditions (3.6 points; P = 0.19), although the seafood/plant protein (2.0 points; P < 0.01) and added sugar (0.8 points; P = 0.04) components were improved following peanut intake. The whole grain component was lower with peanuts compared with LFHC (-2.6 points; P < 0.01). In adults with elevated FPG, peanuts as a nighttime snack (28 g/d) did not affect FPG compared with an isocaloric LFHC snack after 6 wk.This trial was registered at clinicaltrials.gov as NCT03654651.

Sections du résumé

BACKGROUND
The glycemic effects of peanuts are not well studied and no trials have been conducted in adults with elevated fasting plasma glucose (FPG). Furthermore, intake of peanuts as a nighttime snack, an eating occasion affecting FPG, has not been examined.
OBJECTIVES
The aim was to determine the effect of consuming 28 g/d of peanuts as a nighttime snack for 6 wk on glycemic control and cardiovascular disease risk factors, compared with an isocaloric lower fat, higher carbohydrate (LFHC) snack (whole grain crackers and low-fat cheese), in adults with elevated FPG.
METHODS
In a randomized crossover trial, 50 adults (FPG 100 ± 8 mg/dL) consumed dry roasted, unsalted peanuts [164 kcal; 11% energy (E) carbohydrate, 17% E protein, and 73% E fat] or a LFHC snack (164 kcal; 54% E carbohydrate, 17% E protein, and 33% E fat) in the evening (after dinner and before bedtime) for 6 wk with a 4-wk washout period. Primary (FPG) and secondary end points [Healthy Eating Index-2015 (HEI-2015), weight, insulin, fructosamine, lipids/lipoproteins, central and peripheral blood pressure, and pulse wave velocity] were evaluated at the beginning and end of each condition. Linear mixed models were used for data analysis.
RESULTS
FPG was not different between the peanut and LFHC conditions (end point mean difference: -0.6 mg/dL; 95% CI: -2.7, 1.6; P = 0.67). There were no between-condition effects for secondary cardiometabolic endpoints. The HEI-2015 score was not different between the conditions (3.6 points; P = 0.19), although the seafood/plant protein (2.0 points; P < 0.01) and added sugar (0.8 points; P = 0.04) components were improved following peanut intake. The whole grain component was lower with peanuts compared with LFHC (-2.6 points; P < 0.01).
CONCLUSIONS
In adults with elevated FPG, peanuts as a nighttime snack (28 g/d) did not affect FPG compared with an isocaloric LFHC snack after 6 wk.This trial was registered at clinicaltrials.gov as NCT03654651.

Identifiants

pubmed: 34562081
pii: S0022-3166(22)00512-0
doi: 10.1093/jn/nxab347
pmc: PMC8754578
doi:

Substances chimiques

Blood Glucose 0
Glucose IY9XDZ35W2

Banques de données

ClinicalTrials.gov
['NCT03654651']

Types de publication

Journal Article Randomized Controlled Trial Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

153-162

Subventions

Organisme : NCATS NIH HHS
ID : UL1 TR002014
Pays : United States

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press on behalf of the American Society for Nutrition.

Références

PLoS One. 2014 Jul 30;9(7):e103376
pubmed: 25076495
Br J Nutr. 2019 Aug 28;122(4):361-375
pubmed: 31196252
Am J Clin Nutr. 2020 Nov 24;:
pubmed: 33236043
Nutr J. 2013 Mar 27;12:35
pubmed: 23537225
Br J Nutr. 2013 Jun;109(11):2015-23
pubmed: 23122211
Am J Clin Nutr. 2015 Dec;102(6):1347-56
pubmed: 26561616
Endocrinol Metab Clin North Am. 2018 Mar;47(1):33-50
pubmed: 29407055
Diabetes Res Clin Pract. 2001 Sep;53(3):137-9
pubmed: 11483228
Metabolism. 2011 Apr;60(4):474-9
pubmed: 20580779
Lancet. 2010 Jun 26;375(9733):2215-22
pubmed: 20609967
J Nutr. 2017 May;147(5):835-840
pubmed: 28356431
Eur J Clin Nutr. 2010 Aug;64(8):782-91
pubmed: 20502471
Clin Nutr. 2020 Dec;39(12):3601-3606
pubmed: 32204977
J Nutr. 2005 Sep;135(9):2082-9
pubmed: 16140880
Am J Clin Nutr. 2014 Jul;100(1):278-88
pubmed: 24898241
Clin Chem. 2008 Aug;54(8):1307-16
pubmed: 18515257
PLoS One. 2020 Nov 9;15(11):e0241993
pubmed: 33166340
Lancet Diabetes Endocrinol. 2016 Jan;4(1):44-51
pubmed: 26575606
Hypertension. 2019 May;73(5):e35-e66
pubmed: 30827125
Circulation. 2015 Dec 8;132(23):2220-9
pubmed: 26408274
Am J Clin Nutr. 2019 Feb 1;109(2):297-314
pubmed: 30722007
Nutrients. 2015 Sep 02;7(9):7381-98
pubmed: 26404365

Auteurs

Philip A Sapp (PA)

Department of Nutritional Sciences, The Pennsylvania State University, University Park, PA, USA.

Penny M Kris-Etherton (PM)

Department of Nutritional Sciences, The Pennsylvania State University, University Park, PA, USA.

Kristina S Petersen (KS)

Department of Nutritional Sciences, The Pennsylvania State University, University Park, PA, USA.
Department of Nutritional Sciences, Texas Tech University, Lubbock, TX, USA.

Articles similaires

Metabolic engineering of

Jae Sung Cho, Zi Wei Luo, Cheon Woo Moon et al.
1.00
Corynebacterium glutamicum Metabolic Engineering Dicarboxylic Acids Pyridines Pyrones

Glucose and glutamine drive hepatitis E virus replication.

Shaheen Khan, Suruchi Aggarwal, Pooja Bhatia et al.
1.00
Glutamine Virus Replication Hepatitis E virus Glucose Glycolysis

Classifications MeSH