First presentation of a frameshift mutation in the SETD2 gene of a juvenile psammomatoid ossifying fibroma (JPOF) associated with an aneurysmal bone cyst.


Journal

Diagnostic pathology
ISSN: 1746-1596
Titre abrégé: Diagn Pathol
Pays: England
ID NLM: 101251558

Informations de publication

Date de publication:
17 Oct 2021
Historique:
received: 18 06 2021
accepted: 27 09 2021
entrez: 18 10 2021
pubmed: 19 10 2021
medline: 9 2 2022
Statut: epublish

Résumé

The rarity of juvenile psammomatoid ossifying fibroma (JPOF) and lack of cytogenetic studies prompted us to report a novel SETD2 gene mutation in a benign odontogenic tumour. A 21-year-old man presented with a hard, expanded mandibular cortex. Computed tomography revealed multilocular radiopacity in the mandible; this was reconstructed via segmental mandibulectomy using a vascularised iliac crest flap. Based on the clinical and histological findings, we diagnosed JPOF associated with an aneurysmal bone cyst. Microscopically, the solid area was characterised by many rounded or angular ossicles in a cellular fibrous stroma. The stromal cells were spindle-like or stellate. Next-generation sequencing detected a frame shift mutation of the SETD2 gene, while the copy number was normal. Our findings suggest further genetic studies should be performed to assess whether this mutation is related to tumour genesis. .

Sections du résumé

BACKGROUND BACKGROUND
The rarity of juvenile psammomatoid ossifying fibroma (JPOF) and lack of cytogenetic studies prompted us to report a novel SETD2 gene mutation in a benign odontogenic tumour.
CASE PRESENTATION METHODS
A 21-year-old man presented with a hard, expanded mandibular cortex. Computed tomography revealed multilocular radiopacity in the mandible; this was reconstructed via segmental mandibulectomy using a vascularised iliac crest flap. Based on the clinical and histological findings, we diagnosed JPOF associated with an aneurysmal bone cyst. Microscopically, the solid area was characterised by many rounded or angular ossicles in a cellular fibrous stroma. The stromal cells were spindle-like or stellate. Next-generation sequencing detected a frame shift mutation of the SETD2 gene, while the copy number was normal.
CONCLUSIONS CONCLUSIONS
Our findings suggest further genetic studies should be performed to assess whether this mutation is related to tumour genesis. .

Identifiants

pubmed: 34657606
doi: 10.1186/s13000-021-01160-w
pii: 10.1186/s13000-021-01160-w
pmc: PMC8520634
doi:

Substances chimiques

Biomarkers, Tumor 0
Histone-Lysine N-Methyltransferase EC 2.1.1.43
SETD2 protein, human EC 2.1.1.43

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

91

Informations de copyright

© 2021. The Author(s).

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Auteurs

A Toferer (A)

Division of Oral and Maxillofacial Surgery, Medical University of Graz, Auenbruggerplatz 5, 8036, Graz, Austria. astrid.toferer@medunigraz.at.

A Truschnegg (A)

Division of Dental Medicine and Oral Health, Medical University of Graz, Graz, Austria.

K Kashofer (K)

Diagnostic and Research Center for Molecular BioMedicine, Diagnostic and Research Institute of Pathology, Medical University of Graz, Graz, Austria.

C Beham-Schmid (C)

Diagnostic and Research Center for Molecular BioMedicine, Diagnostic and Research Institute of Pathology, Medical University of Graz, Graz, Austria.

A Beham (A)

Medical University of Graz, Neue Stiftingtalstraße 6, 8036, Graz, Austria.

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Classifications MeSH