The role of DNA demethylation in liver to pancreas transdifferentiation.

Cell replacement therapy for diabetes DNA methylation Epigenetic modifications Liver Pancreas Pancreatic transcription factors Transdifferentiation

Journal

Stem cell research & therapy
ISSN: 1757-6512
Titre abrégé: Stem Cell Res Ther
Pays: England
ID NLM: 101527581

Informations de publication

Date de publication:
16 09 2022
Historique:
received: 26 04 2022
accepted: 28 08 2022
entrez: 16 9 2022
pubmed: 17 9 2022
medline: 21 9 2022
Statut: epublish

Résumé

Insulin producing cells generated by liver cell transdifferentiation, could serve as an attractive source for regenerative medicine. The present study assesses the relationship between DNA methylation pTFs induced liver to pancreas transdifferentiation. The transdifferentiation process is associated with DNA demethylation, mainly at gene regulatory sites, and with increased expression of these genes. Active inhibition of DNA methylation promotes the pancreatic transcription factor-induced transdifferentiation process, supporting a causal role for DNA demethylation in this process. Transdifferentiation is associated with global DNA hypomethylation, and with increased expression of specific demethylated genes. A combination of epigenetic modulators may be used to increase chromatin accessibility of the pancreatic transcription factors, thus promoting the efficiency of the developmental process.

Sections du résumé

BACKGROUND
Insulin producing cells generated by liver cell transdifferentiation, could serve as an attractive source for regenerative medicine. The present study assesses the relationship between DNA methylation pTFs induced liver to pancreas transdifferentiation.
RESULTS
The transdifferentiation process is associated with DNA demethylation, mainly at gene regulatory sites, and with increased expression of these genes. Active inhibition of DNA methylation promotes the pancreatic transcription factor-induced transdifferentiation process, supporting a causal role for DNA demethylation in this process.
CONCLUSIONS
Transdifferentiation is associated with global DNA hypomethylation, and with increased expression of specific demethylated genes. A combination of epigenetic modulators may be used to increase chromatin accessibility of the pancreatic transcription factors, thus promoting the efficiency of the developmental process.

Identifiants

pubmed: 36114514
doi: 10.1186/s13287-022-03159-6
pii: 10.1186/s13287-022-03159-6
pmc: PMC9482206
doi:

Substances chimiques

Chromatin 0
Insulins 0
Transcription Factors 0
DNA 9007-49-2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

476

Informations de copyright

© 2022. The Author(s).

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Auteurs

Adi Har-Zahav (A)

Institute of Gastroenterology, Hepatology and Nutrition, Schneider Children's Medical Center of Israel, Petach Tikva, Israel.
Sackler School of Medicine, Felsenstein Medical Research Center, Tel Aviv University, Tel Aviv, Israel.
The Sheba Regenerative Medicine, Stem Cell and Tissue Engineering Center, Sheba Medical Center, 5262100, Tel-Hashomer, Israel.

Daniela Lixandru (D)

University of Medicine and Pharmacy 'Carol Davila', 050474, Bucharest, Romania. daniela.lixandru@umfcd.ro.

David Cheishvili (D)

Department of Oncology, McGill University, Montreal, QC, Canada.
HKG Epitherapeutics, Honk Kong, China.

Ioan Valentin Matei (IV)

Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.
Systems Biology of Aging Group, Institute of Biochemistry of the Romanian Academy, 060031, Bucharest, Romania.

Ioana Raluca Florea (IR)

Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.
Biology and Biochemistry Department, Faculty of Biology, Bucharest University, 050095, București, Romania.

Veronica Madalina Aspritoiu (VM)

Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.
Biology and Biochemistry Department, Faculty of Biology, Bucharest University, 050095, București, Romania.

Inna Blus-Kadosh (I)

Orgenesis Ltd, 7414002, Ness Ziona, Israel.

Irit Meivar-Levy (I)

The Sheba Regenerative Medicine, Stem Cell and Tissue Engineering Center, Sheba Medical Center, 5262100, Tel-Hashomer, Israel.
Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.
Orgenesis Ltd, 7414002, Ness Ziona, Israel.

Andreea Madalina Serban (AM)

University of Medicine and Pharmacy 'Carol Davila', 050474, Bucharest, Romania.
Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.

Irinel Popescu (I)

Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.
Center of Excelence in Translational Medicine, Fundeni Clinical Institute, 022328, Bucharest, Romania.

Moshe Szyf (M)

Department of Oncology, McGill University, Montreal, QC, Canada.
HKG Epitherapeutics, Honk Kong, China.

Sarah Ferber (S)

The Sheba Regenerative Medicine, Stem Cell and Tissue Engineering Center, Sheba Medical Center, 5262100, Tel-Hashomer, Israel.
Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania.
Orgenesis Ltd, 7414002, Ness Ziona, Israel.
Department of Human Genetics, Sackler School of Medicine, Tel Aviv University, 6997801, Tel Aviv, Israel.

Simona Olimpia Dima (SO)

Dia-Cure, Acad. Nicolae Cajal Institute of Medical Scientific Research, Titu Maiorescu University, 040441, Bucharest, Romania. dima.simona@gmail.com.
Center of Excelence in Translational Medicine, Fundeni Clinical Institute, 022328, Bucharest, Romania. dima.simona@gmail.com.

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