Phosphoinositide acyl chain saturation drives CD8


Journal

Nature immunology
ISSN: 1529-2916
Titre abrégé: Nat Immunol
Pays: United States
ID NLM: 100941354

Informations de publication

Date de publication:
03 2023
Historique:
received: 03 05 2022
accepted: 03 01 2023
pubmed: 3 2 2023
medline: 4 3 2023
entrez: 2 2 2023
Statut: ppublish

Résumé

How lipidome changes support CD8

Identifiants

pubmed: 36732424
doi: 10.1038/s41590-023-01419-y
pii: 10.1038/s41590-023-01419-y
pmc: PMC10908374
mid: NIHMS1966246
doi:

Substances chimiques

Phosphatidylinositols 0
Phosphatidylinositol Phosphates 0
Type C Phospholipases EC 3.1.4.-

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

516-530

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : NIAID NIH HHS
ID : R01 AI156274
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM136577
Pays : United States

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Nature America, Inc.

Références

Waickman, A. T. & Powell, J. D. mTOR, metabolism, and the regulation of T cell differentiation and function. Immunol. Rev. 249, 43–58 (2012).
pubmed: 22889214 pmcid: 3419491 doi: 10.1111/j.1600-065X.2012.01152.x
MacIver, N. J., Michalek, R. D. & Rathmell, J. C. Metabolic regulation of T lymphocytes. Annu Rev. Immunol. 31, 259–283 (2013).
pubmed: 23298210 pmcid: 3606674 doi: 10.1146/annurev-immunol-032712-095956
Shyer, J. A., Flavell, R. A. & Bailis, W. Metabolic signaling in T cells. Cell Res 30, 649–659 (2020).
pubmed: 32709897 pmcid: 7395146 doi: 10.1038/s41422-020-0379-5
Carr, E. L. et al. Glutamine uptake and metabolism are coordinately regulated by ERK/MAPK during T lymphocyte activation. J. Immunol. 185, 1037–1044 (2010).
pubmed: 20554958 doi: 10.4049/jimmunol.0903586
Wang, R. et al. The transcription factor Myc controls metabolic reprogramming upon T lymphocyte activation. Immunity 35, 871–882 (2011).
pubmed: 22195744 pmcid: 3248798 doi: 10.1016/j.immuni.2011.09.021
Maciver, N. J. et al. Glucose metabolism in lymphocytes is a regulated process with significant effects on immune cell function and survival. J. Leukoc. Biol. 84, 949–957 (2008).
pubmed: 18577716 pmcid: 2638731 doi: 10.1189/jlb.0108024
Cham, C. M. & Gajewski, T. F. Glucose availability regulates IFN-γ production and p70S6 kinase activation in CD8
pubmed: 15814691 doi: 10.4049/jimmunol.174.8.4670
Barneda, D., Cosulich, S., Stephens, L. & Hawkins, P. How is the acyl chain composition of phosphoinositides created and does it matter? Biochem. Soc. Trans. 47, 1291–1305 (2019).
pubmed: 31657437 pmcid: 6824679 doi: 10.1042/BST20190205
Sasaki, T. et al. Mammalian phosphoinositide kinases and phosphatases. Prog. Lipid Res. 48, 307–343 (2009).
pubmed: 19580826 doi: 10.1016/j.plipres.2009.06.001
Czech, M. P. PIP2 and PIP3: complex roles at the cell surface. Cell 100, 603–606 (2000).
pubmed: 10761925 doi: 10.1016/S0092-8674(00)80696-0
Huang, Y. H. & Sauer, K. Lipid signaling in T cell development and function. Cold Spring Harb. Perspect. Biol. 2, a002428 (2010).
pubmed: 20943760 pmcid: 2964181 doi: 10.1101/cshperspect.a002428
Berridge, M. J. Inositol trisphosphate and calcium signalling mechanisms. Biochim. Biophys. Acta 1793, 933–940 (2009).
pubmed: 19010359 doi: 10.1016/j.bbamcr.2008.10.005
D’Souza, W. N., Chang, C. F., Fischer, A. M., Li, M. & Hedrick, S. M. The Erk2 MAPK regulates CD8 T cell proliferation and survival. J. Immunol. 181, 7617–7629 (2008).
pubmed: 19017950 doi: 10.4049/jimmunol.181.11.7617
Joseph, N., Reicher, B. & Barda-Saad, M. The calcium feedback loop and T cell activation: how cytoskeleton networks control intracellular calcium flux. Biochim. Biophys. Acta 1838, 557–568 (2014).
pubmed: 23860253 doi: 10.1016/j.bbamem.2013.07.009
Sun, Y., Dandekar, R. D., Mao, Y. S., Yin, H. L. & Wulfing, C. Phosphatidylinositol (4,5) bisphosphate controls T cell activation by regulating T cell rigidity and organization. PLoS One 6, e27227 (2011).
pubmed: 22096541 pmcid: 3214035 doi: 10.1371/journal.pone.0027227
Vickers, J. D. & Mustard, J. F. The phosphoinositides exist in multiple metabolic pools in rabbit platelets. Biochem. J. 238, 411–417 (1986).
pubmed: 3026351 pmcid: 1147151 doi: 10.1042/bj2380411
Chakrabarti, P. et al. A dPIP5K dependent pool of phosphatidylinositol 4,5 bisphosphate (PIP2) is required for G-protein-coupled signal transduction in Drosophila photoreceptors. PLoS Genet. 11, e1004948 (2015).
pubmed: 25633995 pmcid: 4310717 doi: 10.1371/journal.pgen.1004948
Fujita, A., Cheng, J., Tauchi-Sato, K., Takenawa, T. & Fujimoto, T. A distinct pool of phosphatidylinositol 4,5-bisphosphate in caveolae revealed by a nanoscale labeling technique. Proc. Natl Acad. Sci. USA 106, 9256–9261 (2009).
pubmed: 19470488 pmcid: 2695096 doi: 10.1073/pnas.0900216106
Costello, P. S., Gallagher, M. & Cantrell, D. A. Sustained and dynamic inositol lipid metabolism inside and outside the immunological synapse. Nat. Immunol. 3, 1082–1089 (2002).
pubmed: 12389042 doi: 10.1038/ni848
Latour, S. & Fischer, A. Signaling pathways involved in the T cell-mediated immunity against Epstein–Barr virus: lessons from genetic diseases. Immunol. Rev. 291, 174–189 (2019).
pubmed: 31402499 doi: 10.1111/imr.12791
Imoto, M., Taniguchi, Y. & Umezawa, K. Inhibition of CDP-DG: inositol transferase by inostamycin. J. Biochem. 112, 299–302 (1992).
pubmed: 1328172 doi: 10.1093/oxfordjournals.jbchem.a123894
Bengsch, B. et al. Analysis of CD127 and KLRG1 expression on hepatitis C virus-specific CD8
pubmed: 17079288 doi: 10.1128/JVI.01354-06
Ward, S. G. & Cantrell, D. A. Phosphoinositide 3-kinases in T lymphocyte activation. Curr. Opin. Immunol. 13, 332–338 (2001).
pubmed: 11406365 doi: 10.1016/S0952-7915(00)00223-5
Hawse, W. F. & Cattley, R. T. T cells transduce T cell receptor signal strength by generating different phosphatidylinositols. J. Biol. Chem. 294, 4793–4805 (2019).
pubmed: 30692200 pmcid: 6442064 doi: 10.1074/jbc.RA118.006524
Hwang, J. R., Byeon, Y., Kim, D. & Park, S. G. Recent insights of T cell receptor-mediated signaling pathways for T cell activation and development. Exp. Mol. Med. 52, 750–761 (2020).
pubmed: 32439954 pmcid: 7272404 doi: 10.1038/s12276-020-0435-8
Janes, P. W., Ley, S. C., Magee, A. I. & Kabouridis, P. S. The role of lipid rafts in T cell antigen receptor (TCR) signalling. Semin. Immunol. 12, 23–34 (2000).
pubmed: 10723795 doi: 10.1006/smim.2000.0204
Holmgren, J., Lonnroth, I. & Svennerholm, L. Tissue receptor for cholera exotoxin: postulated structure from studies with GM1 ganglioside and related glycolipids. Infect. Immun. 8, 208–214 (1973).
pubmed: 4125267 pmcid: 422834 doi: 10.1128/iai.8.2.208-214.1973
Waddington, K. E., Pineda-Torra, I. & Jury, E. C. Analyzing T cell plasma membrane lipids by flow cytometry. Methods Mol. Biol. 1951, 209–216 (2019).
pubmed: 30825155 doi: 10.1007/978-1-4939-9130-3_16
Maib, H. & Murray, D. H. A mechanism for exocyst-mediated tethering via Arf6 and PIP5K1C-driven phosphoinositide conversion. Curr. Biol. 32, 2821–2833 (2022).
pubmed: 35609603 pmcid: 9382030 doi: 10.1016/j.cub.2022.04.089
Zhao, Y., Chen, Y. Q., Li, S., Konrad, R. J. & Cao, G. The microsomal cardiolipin remodeling enzyme acyl-CoA lysocardiolipin acyltransferase is an acyltransferase of multiple anionic lysophospholipids. J. Lipid Res. 50, 945–956 (2009).
pubmed: 19075029 pmcid: 2666181 doi: 10.1194/jlr.M800567-JLR200
Bone, L. N. et al. The acyltransferase LYCAT controls specific phosphoinositides and related membrane traffic. Mol. Biol. Cell 28, 161–172 (2017).
pubmed: 28035047 pmcid: 5221620 doi: 10.1091/mbc.e16-09-0668
Lee, H. C. et al. Caenorhabditis elegans mboa-7, a member of the MBOAT family, is required for selective incorporation of polyunsaturated fatty acids into phosphatidylinositol. Mol. Biol. Cell 19, 1174–1184 (2008).
pubmed: 18094042 pmcid: 2262980 doi: 10.1091/mbc.e07-09-0893
van der Windt, G. J. et al. CD8 memory T cells have a bioenergetic advantage that underlies their rapid recall ability. Proc. Natl Acad. Sci. USA 110, 14336–14341 (2013).
pubmed: 23940348 pmcid: 3761631 doi: 10.1073/pnas.1221740110
Bengsch, B. et al. Bioenergetic insufficiencies due to metabolic alterations regulated by the inhibitory receptor PD-1 are an early driver of CD8
pubmed: 27496729 pmcid: 4988919 doi: 10.1016/j.immuni.2016.07.008
Chang, C. H. et al. Metabolic competition in the tumor microenvironment is a driver of cancer progression. Cell 162, 1229–1241 (2015).
pubmed: 26321679 pmcid: 4864363 doi: 10.1016/j.cell.2015.08.016
Wolchok, J. D. et al. Nivolumab plus ipilimumab in advanced melanoma. N. Engl. J. Med. 369, 122–133 (2013).
pubmed: 23724867 pmcid: 5698004 doi: 10.1056/NEJMoa1302369
Hawse, W. F., Boggess, W. C. & Morel, P. A. TCR signal strength regulates Akt substrate specificity to induce alternate murine T
pubmed: 28600288 doi: 10.4049/jimmunol.1700369
Lee, K. H. et al. T cell receptor signaling precedes immunological synapse formation. Science 295, 1539–1542 (2002).
pubmed: 11859198 doi: 10.1126/science.1067710
Lee, K. H. et al. The immunological synapse balances T cell receptor signaling and degradation. Science 302, 1218–1222 (2003).
pubmed: 14512504 doi: 10.1126/science.1086507
Traynor-Kaplan, A. et al. Fatty-acyl chain profiles of cellular phosphoinositides. Biochim. Biophys. Acta Mol. Cell Biol. Lipids 1862, 513–522 (2017).
pubmed: 28189644 doi: 10.1016/j.bbalip.2017.02.002
Naguib, A. et al. p53 mutations change phosphatidylinositol acyl chain composition. Cell Rep. 10, 8–19 (2015).
pubmed: 25543136 doi: 10.1016/j.celrep.2014.12.010
van der Windt, G. J. & Pearce, E. L. Metabolic switching and fuel choice during T cell differentiation and memory development. Immunol. Rev. 249, 27–42 (2012).
pubmed: 22889213 pmcid: 3645891 doi: 10.1111/j.1600-065X.2012.01150.x
Frauwirth, K. A. et al. The CD28 signaling pathway regulates glucose metabolism. Immunity 16, 769–777 (2002).
pubmed: 12121659 doi: 10.1016/S1074-7613(02)00323-0
Ho, P. C. et al. Phosphoenolpyruvate is a metabolic checkpoint of anti-tumor T cell responses. Cell 162, 1217–1228 (2015).
pubmed: 26321681 pmcid: 4567953 doi: 10.1016/j.cell.2015.08.012
Barneda, D. et al. Acyl chain selection couples the consumption and synthesis of phosphoinositides. EMBO J. 41, e110038 (2022).
pubmed: 35771169 pmcid: 9475507 doi: 10.15252/embj.2021110038
Shulga, Y. V., Anderson, R. A., Topham, M. K. & Epand, R. M. Phosphatidylinositol-4-phosphate 5-kinase isoforms exhibit acyl chain selectivity for both substrate and lipid activator. J. Biol. Chem. 287, 35953–35963 (2012).
pubmed: 22942276 pmcid: 3476263 doi: 10.1074/jbc.M112.370155
Blunsom, N. J. & Cockcroft, S. Phosphatidylinositol synthesis at the endoplasmic reticulum. Biochim. Biophys. Acta Mol. Cel. Biol. Lipids 1865, 158471 (2020).
doi: 10.1016/j.bbalip.2019.05.015
D’Souza, K. & Epand, R. M. The phosphatidylinositol synthase-catalyzed formation of phosphatidylinositol does not exhibit acyl chain specificity. Biochemistry 54, 1151–1153 (2015).
pubmed: 25633188 doi: 10.1021/bi5015634
Pike, L. J. Lipid rafts: bringing order to chaos. J. Lipid Res. 44, 655–667 (2003).
pubmed: 12562849 doi: 10.1194/jlr.R200021-JLR200
Myeong, J., Park, C. G., Suh, B. C. & Hille, B. Compartmentalization of phosphatidylinositol 4,5-bisphosphate metabolism into plasma membrane liquid-ordered/raft domains. Proc. Natl Acad. Sci. USA 118, e2025343118 (2021).
pubmed: 33619111 pmcid: 7936302 doi: 10.1073/pnas.2025343118
Veri, M. C. et al. Membrane raft-dependent regulation of phospholipase Cγ-1 activation in T lymphocytes. Mol. Cell. Biol. 21, 6939–6950 (2001).
pubmed: 11564877 pmcid: 99870 doi: 10.1128/MCB.21.20.6939-6950.2001
Kallikourdis, M. et al. Phosphatidylinositol 4-phosphate 5-kinase beta controls recruitment of lipid rafts into the immunological synapse. J. Immunol. 196, 1955–1963 (2016).
pubmed: 26773155 doi: 10.4049/jimmunol.1501788
Parry, R. V. et al. CTLA-4 and PD-1 receptors inhibit T cell activation by distinct mechanisms. Mol. Cell. Biol. 25, 9543–9553 (2005).
pubmed: 16227604 pmcid: 1265804 doi: 10.1128/MCB.25.21.9543-9553.2005
Callahan, M. K., Postow, M. A. & Wolchok, J. D. Targeting T cell co-receptors for cancer therapy. Immunity 44, 1069–1078 (2016).
pubmed: 27192570 doi: 10.1016/j.immuni.2016.04.023
Matyash, V., Liebisch, G., Kurzchalia, T. V., Shevchenko, A. & Schwudke, D. Lipid extraction by methyl-tert-butyl ether for high-throughput lipidomics. J. Lipid Res. 49, 1137–1146 (2008).
pubmed: 18281723 pmcid: 2311442 doi: 10.1194/jlr.D700041-JLR200
Wong, M., Xu, G., Park, D., Barboza, M. & Lebrilla, C. B. Intact glycosphingolipidomic analysis of the cell membrane during differentiation yields extensive glycan and lipid changes. Sci. Rep. 8, 10993 (2018).
pubmed: 30030471 pmcid: 6054638 doi: 10.1038/s41598-018-29324-7
Clark, J. et al. Quantification of PtdInsP3 molecular species in cells and tissues by mass spectrometry. Nat. Methods 8, 267–272 (2011).
pubmed: 21278744 pmcid: 3460242 doi: 10.1038/nmeth.1564
Wills, J., Edwards-Hicks, J. & Finch, A. J. AssayR: a simple mass spectrometry software tool for targeted metabolic and stable isotope tracer analyses. Anal. Chem. 89, 9616–9619 (2017).
pubmed: 28850215 pmcid: 5628912 doi: 10.1021/acs.analchem.7b02401
O’Sullivan, D. et al. Fever supports CD8
pubmed: 34161266 pmcid: 8237659 doi: 10.1073/pnas.2023752118
Bolstad, B. preprocessCore: a collection of pre-processing functions. R package version 1.42.0. https://github.com/bmbolstad/preprocessCore

Auteurs

Joy Edwards-Hicks (J)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Petya Apostolova (P)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Joerg M Buescher (JM)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Hannes Maib (H)

Division of Cell & Developmental Biology, School of Life Sciences, University of Dundee, Dundee, UK.

Michal A Stanczak (MA)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Mauro Corrado (M)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Ramon I Klein Geltink (RI)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Maria Elena Maccari (ME)

Center for Chronic Immunodeficiency, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Matteo Villa (M)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Gustavo E Carrizo (GE)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

David E Sanin (DE)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Francesc Baixauli (F)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Beth Kelly (B)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Jonathan D Curtis (JD)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Fabian Haessler (F)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Annette Patterson (A)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Cameron S Field (CS)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

George Caputa (G)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Ryan L Kyle (RL)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Melanie Soballa (M)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Minsun Cha (M)

Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Harry Paul (H)

Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Jacob Martin (J)

Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Katarzyna M Grzes (KM)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Lea Flachsmann (L)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Michael Mitterer (M)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Liang Zhao (L)

Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

Frances Winkler (F)

Clinic for Internal Medicine II, Gastroenterology, Hepatology, Endocrinology, and Infectious Diseases, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.
Faculty of Biology, University of Freiburg, Freiburg, Germany.

David Ali Rafei-Shamsabadi (DA)

Department of Dermatology, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Frank Meiss (F)

Department of Dermatology, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Bertram Bengsch (B)

Clinic for Internal Medicine II, Gastroenterology, Hepatology, Endocrinology, and Infectious Diseases, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.
Signaling Research Centers BIOSS and CIBSS, University of Freiburg, Freiburg, Germany.

Robert Zeiser (R)

Signaling Research Centers BIOSS and CIBSS, University of Freiburg, Freiburg, Germany.
Department of Medicine I, Medical Center - University of Freiburg, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Daniel J Puleston (DJ)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.

David O'Sullivan (D)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.

Edward J Pearce (EJ)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA.
Faculty of Biology, University of Freiburg, Freiburg, Germany.

Erika L Pearce (EL)

Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Germany. epearce6@jhmi.edu.
Bloomberg-Kimmel Institute for Cancer Immunotherapy and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD, USA. epearce6@jhmi.edu.

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