Utility of genetic testing in children with leukodystrophy.


Journal

European journal of paediatric neurology : EJPN : official journal of the European Paediatric Neurology Society
ISSN: 1532-2130
Titre abrégé: Eur J Paediatr Neurol
Pays: England
ID NLM: 9715169

Informations de publication

Date de publication:
Jul 2023
Historique:
received: 04 02 2023
accepted: 22 05 2023
medline: 24 7 2023
pubmed: 3 6 2023
entrez: 2 6 2023
Statut: ppublish

Résumé

Leukodystrophies are monogenic disorders primarily affecting the white matter. We aimed to evaluate the utility of genetic testing and time-to-diagnosis in a retrospective cohort of children with suspected leukodystrophy. Medical records of patients who attended the leukodystrophy clinic at the Dana-Dwek Children's Hospital between June 2019 and December 2021 were retrieved. Clinical, molecular, and neuroimaging data were reviewed, and the diagnostic yield was compared across genetic tests. Sixty-seven patients (Female/Male ratio 35/32) were included. Median age at symptom onset was 9 months (interquartile range (IQR) 3-18 months), and median length of follow-up was 4.75 years (IQR 3-8.5). Time from symptom onset to a confirmed genetic diagnosis was 15months (IQR 11-30). Pathogenic variants were identified in 60/67 (89.6%) patients; classic leukodystrophy (55/67, 82.1%), leukodystrophy mimics (5/67, 7.5%). Seven patients (10.4%) remained undiagnosed. Exome sequencing showed the highest diagnostic yield (34/41, 82.9%), followed by single-gene sequencing (13/24, 54%), targeted panels (3/9, 33.3%) and chromosomal microarray (2/25, 8%). Familial pathogenic variant testing confirmed the diagnosis in 7/7 patients. A comparison between patients who presented before (n = 31) and after (n = 21) next-generation sequencing (NGS) became clinically available in Israel revealed that the time-to-diagnosis was shorter in the latter group with a median of 12months (IQR 3.5-18.5) vs. a median of 19 months (IQR 13-51) (p = 0.005). NGS carries the highest diagnostic yield in children with suspected leukodystrophy. Access to advanced sequencing technologies accelerates speed to diagnosis, which is increasingly crucial as targeted treatments become available.

Sections du résumé

BACKGROUND BACKGROUND
Leukodystrophies are monogenic disorders primarily affecting the white matter. We aimed to evaluate the utility of genetic testing and time-to-diagnosis in a retrospective cohort of children with suspected leukodystrophy.
METHODS METHODS
Medical records of patients who attended the leukodystrophy clinic at the Dana-Dwek Children's Hospital between June 2019 and December 2021 were retrieved. Clinical, molecular, and neuroimaging data were reviewed, and the diagnostic yield was compared across genetic tests.
RESULTS RESULTS
Sixty-seven patients (Female/Male ratio 35/32) were included. Median age at symptom onset was 9 months (interquartile range (IQR) 3-18 months), and median length of follow-up was 4.75 years (IQR 3-8.5). Time from symptom onset to a confirmed genetic diagnosis was 15months (IQR 11-30). Pathogenic variants were identified in 60/67 (89.6%) patients; classic leukodystrophy (55/67, 82.1%), leukodystrophy mimics (5/67, 7.5%). Seven patients (10.4%) remained undiagnosed. Exome sequencing showed the highest diagnostic yield (34/41, 82.9%), followed by single-gene sequencing (13/24, 54%), targeted panels (3/9, 33.3%) and chromosomal microarray (2/25, 8%). Familial pathogenic variant testing confirmed the diagnosis in 7/7 patients. A comparison between patients who presented before (n = 31) and after (n = 21) next-generation sequencing (NGS) became clinically available in Israel revealed that the time-to-diagnosis was shorter in the latter group with a median of 12months (IQR 3.5-18.5) vs. a median of 19 months (IQR 13-51) (p = 0.005).
CONCLUSIONS CONCLUSIONS
NGS carries the highest diagnostic yield in children with suspected leukodystrophy. Access to advanced sequencing technologies accelerates speed to diagnosis, which is increasingly crucial as targeted treatments become available.

Identifiants

pubmed: 37267771
pii: S1090-3798(23)00087-9
doi: 10.1016/j.ejpn.2023.05.008
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

29-35

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2023 Published by Elsevier Ltd on behalf of European Paediatric Neurology Society.

Déclaration de conflit d'intérêts

Declaration of competing interest We declare no competing interests.

Auteurs

Ayelet Zerem (A)

Pediatric Neurology Institute, Dana-Dwek Children's Hospital, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel; Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel. Electronic address: ayeletze@tlvmc.gov.il.

Stephanie Libzon (S)

Pediatric Neurology Institute, Dana-Dwek Children's Hospital, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel; Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel; Magen Rare Disease Center, Pediatric Neurology Unit, Wolfson Medical Center, Holon, Israel.

Liat Ben Sira (L)

Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel; Pediatric Radiology, Department of Radiology, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel.

Hadas Meirson (H)

Pediatric Neurology Institute, Dana-Dwek Children's Hospital, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel; Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Moran Hausman-Kedem (M)

Pediatric Neurology Institute, Dana-Dwek Children's Hospital, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel; Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Noam Haviv (N)

Statistical Advisor and Senior Lecturer, The Ashkelon Academic College, Israel.

Keren Yosovich (K)

Magen Rare Disease Center, Genetics Institute, Wolfson Medical Center, Holon, Israel.

Adi Mory (A)

Genetics Institute and Genomic Center, Tel Aviv Sourasky Medical Center, Israel.

Hagit Baris Feldman (H)

Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel; Genetics Institute and Genomic Center, Tel Aviv Sourasky Medical Center, Israel.

Dorit Lev (D)

Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel; Magen Rare Disease Center, Genetics Institute, Wolfson Medical Center, Holon, Israel.

Tally Lerman-Sagie (T)

Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel; Magen Rare Disease Center, Pediatric Neurology Unit, Wolfson Medical Center, Holon, Israel.

Aviva Fattal-Valevski (A)

Pediatric Neurology Institute, Dana-Dwek Children's Hospital, Tel Aviv Sourasky Medical Center, Tel Aviv, Israel; Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Yael Hacohen (Y)

Queen Square MS Centre, UCL Queen Square Institute of Neurology, Faculty of Brain Sciences, University College London, London, United Kingdom; Department of Neurology, Great Ormond Street Hospital for Children, London, United Kingdom.

Daphna Marom (D)

Sackler Faulty of Medicine, Tel Aviv University, Tel Aviv, Israel; Genetics Institute and Genomic Center, Tel Aviv Sourasky Medical Center, Israel.

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