Serotonin reuptake inhibitors improve muscle stem cell function and muscle regeneration in male mice.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
31 Jul 2024
Historique:
received: 16 06 2021
accepted: 03 07 2024
medline: 1 8 2024
pubmed: 1 8 2024
entrez: 31 7 2024
Statut: epublish

Résumé

Serotonin reuptake inhibitor antidepressants such as fluoxetine are widely used to treat mood disorders. The mechanisms of action include an increase in extracellular level of serotonin, neurogenesis, and growth of vessels in the brain. We investigated whether fluoxetine could have broader peripheral regenerative properties. Following prolonged administration of fluoxetine in male mice, we showed that fluoxetine increases the number of muscle stem cells and muscle angiogenesis, associated with positive changes in skeletal muscle function. Fluoxetine also improved skeletal muscle regeneration after single and multiples injuries with an increased muscle stem cells pool and vessel density associated with reduced fibrotic lesions and inflammation. Mice devoid of peripheral serotonin treated with fluoxetine did not exhibit beneficial effects during muscle regeneration. Specifically, pharmacological, and genetic inactivation of the 5-HT1B subtype serotonin receptor also abolished the enhanced regenerative process induced by fluoxetine. We highlight here a regenerative property of serotonin on skeletal muscle.

Identifiants

pubmed: 39085209
doi: 10.1038/s41467-024-50220-4
pii: 10.1038/s41467-024-50220-4
doi:

Substances chimiques

Selective Serotonin Reuptake Inhibitors 0
Fluoxetine 01K63SUP8D
Serotonin 333DO1RDJY

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6457

Informations de copyright

© 2024. The Author(s).

Références

Wong, D. T., Horng, J. S., Bymaster, F. P., Hauser, K. L. & Molloy, B. B. A selective inhibitor of serotonin uptake: Lilly 110140, 3-(p-Trifluoromethylphenoxy)-n-methyl-3-phenylpropylamine. Life Sci. 15, 471–479 (1974).
pubmed: 4549929 doi: 10.1016/0024-3205(74)90345-2
Wong, D. T., Perry, K. W. & Bymaster, F. P. Case history: the discovery of fluoxetine hydrochloride (Prozac). Nat. Rev. Drug Discov. 4, 764–774 (2005).
pubmed: 16121130 doi: 10.1038/nrd1821
Fritze, S., Spanagel, R. & Noori, H. R. Adaptive dynamics of the 5-HT systems following chronic administration of selective serotonin reuptake inhibitors: a meta-analysis. J. Neurochem. 142, 747–755 (2017).
pubmed: 28653748 doi: 10.1111/jnc.14114
Bäckman, J., Alling, C., Alsén, M., Regnéll, G. & Träskman-Bendz, L. Changes of cerebrospinal fluid monoamine metabolites during long-term antidepressant treatment. Eur. Neuropsychopharmacol. 10, 341–349 (2000).
pubmed: 10974605 doi: 10.1016/S0924-977X(00)00089-4
Encinas, J. M., Vaahtokari, A. & Enikolopov, G. Fluoxetine targets early progenitor cells in the adult brain. Proc. Natl. Acad. Sci. 103, 8233–8238 (2006).
pubmed: 16702546 pmcid: 1461404 doi: 10.1073/pnas.0601992103
Malberg, J. E., Eisch, A. J., Nestler, E. J. & Duman, R. S. Chronic antidepressant treatment increases neurogenesis in adult rat hippocampus. J. Neurosci. 20, 9104–9110 (2000).
pubmed: 11124987 pmcid: 6773038 doi: 10.1523/JNEUROSCI.20-24-09104.2000
Santarelli, L. et al. Requirement of hippocampal neurogenesis for the behavioral effects of antidepressants. Science (1979) 301, 805–809 (2003).
Wang, J. W., David, D. J., Monckton, J. E., Battaglia, F. & Hen, R. Chronic fluoxetine stimulates maturation and synaptic plasticity of adult-born hippocampal granule cells. J. Neurosci. 28, 1374–1384 (2008).
pubmed: 18256257 pmcid: 6671574 doi: 10.1523/JNEUROSCI.3632-07.2008
Warner-Schmidt, J. L. & Duman, R. S. VEGF is an essential mediator of the neurogenic and behavioral actions of antidepressants. Proc. Natl Acad. Sci. 104, 4647–4652 (2007).
pubmed: 17360578 pmcid: 1838655 doi: 10.1073/pnas.0610282104
Lee, J. S. et al. Induction of neuronal vascular endothelial growth factor expression by cAMP in the dentate gyrus of the hippocampus is required for antidepressant-like behaviors. J. Neurosci. 29, 8493–8505 (2009).
pubmed: 19571140 pmcid: 2796224 doi: 10.1523/JNEUROSCI.1321-09.2009
Amireault, P., Sibon, D. & Coîté, F. Life without peripheral serotonin: Insights from tryptophan hydroxylase 1 knockout mice reveal the existence of paracrine/autocrine serotonergic networks. ACS Chem. Neurosci. 4, 64–71 (2013).
pubmed: 23336045 doi: 10.1021/cn300154j
Lesurtel, M. Platelet-derived serotonin mediates liver regeneration. Science (1979) 312, 104–107 (2006).
Baudry, A. et al. Essential roles of dopamine and serotonin in tooth repair: Functional interplay between odontogenic stem cells and platelets. Stem Cells 33, 2586–2595 (2015).
pubmed: 25865138 doi: 10.1002/stem.2037
Gayraud-Morel, B., Chrétien, F. & Tajbakhsh, S. Skeletal muscle as a paradigm for regenerative biology and medicine. Regenerative Med. 4, 293–319 (2009).
doi: 10.2217/17460751.4.2.293
Sambasivan, R. et al. Distinct regulatory cascades govern extraocular and pharyngeal arch muscle progenitor cell fates. Dev. Cell 16, 810–821 (2009).
pubmed: 19531352 doi: 10.1016/j.devcel.2009.05.008
Ema, M., Takahashi, S. & Rossant, J. Deletion of the selection cassette, but not cis-acting elements, in targeted Flk1-lacZ allele reveals Flk1 expression in multipotent mesodermal progenitors. Blood 107, 111–117 (2006).
pubmed: 16166582 doi: 10.1182/blood-2005-05-1970
Malinda, K. M. In vivo matrigel migration and angiogenesis assay. Methods Mol. Biol. 467, 287–294 (2009).
pubmed: 19301678 doi: 10.1007/978-1-59745-241-0_17
Hardy, D. et al. Comparative Study of Injury Models for Studying Muscle Regeneration in Mice. PLOS ONE 11, e0147198 (2016).
pubmed: 26807982 pmcid: 4726569 doi: 10.1371/journal.pone.0147198
Hajduch, E. et al. Serotonin (5-hydroxytryptamine), a novel regulator of glucose transport in rat skeletal muscle. J. Biol. Chem. 274, 13563–13568 (1999).
pubmed: 10224126 doi: 10.1074/jbc.274.19.13563
Barnes, N. M. et al. International union of basic and clinical pharmacology. Cx. Classification of receptors for 5-hydroxytryptamine; pharmacology and function. Pharmacol. Rev. 73, 310–520 (2021).
pubmed: 33370241 pmcid: 7770494 doi: 10.1124/pr.118.015552
Cote, F. et al. Disruption of the nonneuronal tph1 gene demonstrates the importance of peripheral serotonin in cardiac function. Proc. Natl Acad. Sci. 100, 13525–13530 (2003).
pubmed: 14597720 pmcid: 263847 doi: 10.1073/pnas.2233056100
Nautiyal, K. M. et al. Distinct circuits underlie the effects of 5-HT1B receptors on aggression and impulsivity. Neuron 86, 813–827 (2015).
pubmed: 25892302 pmcid: 4431594 doi: 10.1016/j.neuron.2015.03.041
Mourikis, P. et al. A critical requirement for notch signaling in maintenance of the quiescent skeletal muscle stem cell state. Stem Cells 30, 243–252 (2012).
pubmed: 22069237 doi: 10.1002/stem.775
Mason, C. & Dunnill, P. A brief definition of regenerative medicine. Regenerative Med. 3, 1–5 (2008).
Yin, H., Price, F. & Rudnicki, M. A. Satellite cells and the muscle stem cell niche. Physiol. Rev. 93, 23–67 (2013).
pubmed: 23303905 pmcid: 4073943 doi: 10.1152/physrev.00043.2011
Pakala, R., Willerson, J. T. & Benedict, C. R. Effect of serotonin, thromboxane A2, and specific receptor antagonists on vascular smooth muscle cell proliferation. Circulation 96, 2280–2286 (1997).
pubmed: 9337201 doi: 10.1161/01.CIR.96.7.2280
Pakala, R. & Benedict, C. R. Effect of serotonin and thromboxane A2 on endothelial cell proliferation: Effect of specific receptor antagonists. J. Lab. Clin. Med. 131, 527–537 (1998).
pubmed: 9626988 doi: 10.1016/S0022-2143(98)90061-0
Balasubramanian, S. & Paulose, C. S. Induction of DNA synthesis in primary cultures of rat hepatocytes by serotonin: Possible involvement of serotonin S2 receptor. Hepatology 27, 62–66 (1998).
pubmed: 9425918 doi: 10.1002/hep.510270111
Chandran, S. et al. Effects of serotonin on skeletal muscle growth. BMC Proc. 6, O3 (2012).
pmcid: 3394452 doi: 10.1186/1753-6561-6-S3-O3
Manoli, I. et al. Monoamine oxidase‐A is a major target gene for glucocorticoids in human skeletal muscle cells. FASEB J. 19, 1359–1361 (2005).
pubmed: 15946989 doi: 10.1096/fj.04-3660fje
Al-Zoairy, R. et al. Serotonin improves glucose metabolism by Serotonylation of the small GTPase Rab4 in L6 skeletal muscle cells. Diabetol. Metab. Syndr. 9, 1–9 (2017).
pubmed: 28053672 pmcid: 5209910 doi: 10.1186/s13098-016-0201-1
Guillet-Deniau, I., Burnol, A. F. & Girard, J. Identification and localization of a skeletal muscle secrotonin 5-HT2A receptor coupled to the Jak/STAT pathway. J. Biol. Chem. 272, 14825–14829 (1997).
pubmed: 9169451 doi: 10.1074/jbc.272.23.14825
Popa, D. et al. A longitudinal study of 5-HT outflow during chronic fluoxetine treatment using a new technique of chronic microdialysis in a highly emotional mouse strain. Eur. J. Pharmacol. 628, 83–90 (2010).
pubmed: 19944680 doi: 10.1016/j.ejphar.2009.11.037
Crane, J. D. et al. Inhibiting peripheral serotonin synthesis reduces obesity and metabolic dysfunction by promoting brown adipose tissue thermogenesis. Nat. Med. 21, 166–172 (2015).
pubmed: 25485911 doi: 10.1038/nm.3766
Yadav, V. K. et al. Lrp5 controls bone formation by inhibiting serotonin synthesis in the duodenum: an entero-bone endocrine axis. Cell 135, 825–837 (2009).
doi: 10.1016/j.cell.2008.09.059
Yang, M. et al. Promoting effects of serotonin on hematopoiesis: ex vivo expansion of cord blood CD34
pubmed: 17446559 doi: 10.1634/stemcells.2007-0048
Amireault, P. et al. Ineffective erythropoiesis with reduced red blood cell survival in serotonin-deficient mice. Proc. Natl Acad. Sci. 108, 13141–13146 (2011).
pubmed: 21788492 pmcid: 3156216 doi: 10.1073/pnas.1103964108
Sibon, D. et al. Enhanced renewal of erythroid progenitors in myelodysplastic anemia by peripheral serotonin. Cell Rep. 26, 3246–3256.e4 (2019).
pubmed: 30893598 doi: 10.1016/j.celrep.2019.02.071
Yang, M., Srikiatkhachorn, A., Anthony, M. & Chong, B. H. Serotonin stimulates megakaryocytopoiesis via the 5-HT2receptor. Blood Coagul. Fibrinolysis 7, 127–133 (1996).
pubmed: 8735801 doi: 10.1097/00001721-199603000-00004
Ye, J. Y. et al. Serotonin enhances megakaryopoiesis and proplatelet formation via p-Erk1/2 and F-actin reorganization. Stem Cells 32, 2973–2982 (2014).
pubmed: 24980849 doi: 10.1002/stem.1777
Chabbi-Achengli, Y. et al. Decreased osteoclastogenesis in serotonin-deficient mice. Proc. Natl. Acad. Sci. USA 109, 2567–2572 (2012).
pubmed: 22308416 pmcid: 3289318 doi: 10.1073/pnas.1117792109
Yadav, V. K. et al. A serotonin-dependent mechanism explains the leptin regulation of bone mass, appetite, and energy expenditure. Cell 138, 976–989 (2009).
pubmed: 19737523 pmcid: 2768582 doi: 10.1016/j.cell.2009.06.051
Sadiq, A. et al. The role of serotonin during skin healing in post-thermal injury. Int. J. Mol. Sci. 19, 1–20 (2018).
doi: 10.3390/ijms19041034
Furrer, K. et al. Serotonin reverts age-related capillarization and failure of regeneration in the liver through a VEGF-dependent pathway. Proc. Natl Acad. Sci. 108, 2945–2950 (2011).
pubmed: 21282654 pmcid: 3041135 doi: 10.1073/pnas.1012531108
Coman, T. et al. Serotonin targeting using common antidepressants induces rapid recovery of cytopenia. Blood 134, 3715–3715 (2019).
doi: 10.1182/blood-2019-122658
Raymond, J. R. et al. Multiplicity of mechanisms of serotonin receptor signal transduction. Pharmacol. Ther. 92, 179–212 (2001).
Azmitia, E. C. Modern views on an ancient chemical: Serotonin effects on cell proliferation, maturation, and apoptosis. in Brain Research Bulletin vol. 56 413–424 (Brain Res Bull, 2001).
Launay, J. M. et al. Serotonin 5-HT 2B receptors are required for bone-marrow contribution to pulmonary arterial hypertension. Blood 119, 1772–1780 (2012).
pubmed: 22186990 doi: 10.1182/blood-2011-06-358374
Kellermann, O., Loric, S., Maroteaux, L. & Launay, J. M. Sequential onset of three 5-HT receptors during the 5-hydroxytryptaminergic differentiation of the murine 1C11 cell line. Br. J. Pharmacol. 118, 1161–1170 (1996).
pubmed: 8818339 pmcid: 1909597 doi: 10.1111/j.1476-5381.1996.tb15519.x
Tomasetti, C. & Vogelstein, B. Variation in cancer risk among tissues can be explained by the number of stem cell divisions. Science (1979) 347, 78–81 (2015).
Savelieva, K. V. et al. Genetic disruption of both tryptophan hydroxylase genes dramatically reduces serotonin and affects behavior in models sensitive to antidepressants. PLoS ONE 3, (2008).
Kuang, S., Kuroda, K., Le Grand, F. & Rudnicki, M. A. Asymmetric Self-Renewal and Commitment of Satellite Stem Cells in Muscle. Cell 129, 999–1010 (2007).
pubmed: 17540178 pmcid: 2718740 doi: 10.1016/j.cell.2007.03.044
Abou-Khalil, R., Mounier, R. & Chazaud, B. Regulation of myogenic stem cell behavior by vessel cells: The “ménage à trois” of satellite cells, periendothelial cells and endothelial cells. Cell Cycle 9, 892–896 (2010).
Chiristov, C. et al. Muscle satellite cells and endothelial cells: Close neighbors and privileged partners. Mol. Biol. Cell 18, 1397–1409 (2007).
doi: 10.1091/mbc.e06-08-0693
Greene, J., Banasr, M., Lee, B., Warner-Schmidt, J. & Duman, R. S. Vascular endothelial growth factor signaling is required for the behavioral actions of antidepressant treatment: Pharmacological and cellular characterization. Neuropsychopharmacology 34, 2459–2468 (2009).
pubmed: 19553916 doi: 10.1038/npp.2009.68
Fournier, N. M., Lee, B., Banasr, M., Elsayed, M. & Duman, R. S. Vascular endothelial growth factor regulates adult hippocampal cell proliferation through MEK/ERK- and PI3K/Akt-dependent signaling. Neuropharmacology 63, 642–652 (2012).
pubmed: 22580375 pmcid: 3392414 doi: 10.1016/j.neuropharm.2012.04.033
Machida, T., Iizuka, K. & Hirafuji, M. 5-hydroxytryptamine and its receptors in systemic vascular walls. Biol. Pharm. Bull. 36, 1416–1419 (2013).
pubmed: 23995652 doi: 10.1248/bpb.b13-00344
Iwabayashi, M. et al. Role of serotonin in angiogenesis: Induction of angiogenesis by sarpogrelate via endothelial 5-HT1B/Akt/eNOS pathway in diabetic mice. Atherosclerosis 220, 337–342 (2012).
pubmed: 22172591 doi: 10.1016/j.atherosclerosis.2011.10.042
Tidball, J. G. Regulation of muscle growth and regeneration by the immune system. Nat. Rev. Immunol. 17, 165–178 (2017).
Arreola, R. et al. Immunomodulatory effects mediated by serotonin. J. Immunol. Res. 354957 https://doi.org/10.1155/2015/354957 (2015).
de las Casas-Engel, M. et al. Serotonin Skews Human Macrophage Polarization through HTR 2B and HTR 7. J. Immunol. 190, 2301–2310 (2013).
doi: 10.4049/jimmunol.1201133
Dees, C. et al. Platelet-derived serotonin links vascular disease and tissue fibrosis. J. Exp. Med. 208, 961–972 (2011).
pubmed: 21518801 pmcid: 3092343 doi: 10.1084/jem.20101629
Tutakhail, A., Nazari, Q. A., Khabil, S., Gardier, A. & Coudore, F. Muscular and mitochondrial effects of long-term fluoxetine treatment in mice, combined with physical endurance exercise on treadmill. Life Sci. 232, 116508 (2019).
Falabrègue, M. et al. Lack of skeletal muscle serotonin impairs physical performance. Int. J. Tryptophan Res. 14 https://doi.org/10.1177/11786469211003109 (2021).
Carre-Pierrat, M. et al. Pre-clinical study of 21 approved drugs in the mdx mouse. Neuromuscul. Disord. 21, 313–327 (2011).
pubmed: 21392993 doi: 10.1016/j.nmd.2011.01.005
Carre-Pierrat, M. et al. Blocking of striated muscle degeneration by serotonin in C. elegans. J. Muscle Res. Cell Motil. 27, 253–258 (2006).
pubmed: 16791712 doi: 10.1007/s10974-006-9070-9
Waugh, T. A. et al. Fluoxetine prevents dystrophic changes in a zebrafish model of duchenne muscular dystrophy. Hum. Mol. Genet. 23, 4651–4662 (2014).
pubmed: 24760771 pmcid: 4119416 doi: 10.1093/hmg/ddu185
Latil, M. et al. Skeletal muscle stem cells adopt a dormant cell state post mortem and retain regenerative capacity. Nat. Commun. 3, 1–12 (2012).
doi: 10.1038/ncomms1890
Goetsch, K. P., Snyman, C., Myburgh, K. H. & Niesler, C. U. Simultaneous isolation of enriched myoblasts and fibroblasts for migration analysis within a novel co-culture assay. Biotechniques 58, 25–32 (2015).
pubmed: 25605577 doi: 10.2144/000114246
Mayeuf-Louchart, A. et al. MuscleJ: A high-content analysis method to study skeletal muscle with a new Fiji tool. Skelet. Muscle 8, 1–11 (2018).
doi: 10.1186/s13395-018-0171-0

Auteurs

Mylène Fefeu (M)

GHU Paris Psychiatrie & Neurosciences, site Sainte Anne, Service Hospitalo-Universitaire de psychiatrie, Paris, France.
Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France.
Université de Paris Cité, Paris, France.

Michael Blatzer (M)

Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France.

Anita Kneppers (A)

Institut NeuroMyoGène, Unité Physiopathologie et Génétique du Neurone et du Muscle, Université Claude Bernard Lyon 1, CNRS UMR 5261, Inserm U1315, Univ Lyon, Lyon, France.

David Briand (D)

Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France.

Pierre Rocheteau (P)

Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France.

Alexandre Haroche (A)

GHU Paris Psychiatrie & Neurosciences, site Sainte Anne, Service Hospitalo-Universitaire de psychiatrie, Paris, France.

David Hardy (D)

Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France.

Mélanie Juchet-Martin (M)

Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France.

Anne Danckaert (A)

Institut Pasteur, UTechS PBI, C2RT, Paris, France.

François Coudoré (F)

CESP, MOODS Team, Inserm, Faculté de Pharmacie, Université Paris-Saclay, Châtenay-Malabry, France.

Abdulkarim Tutakhail (A)

CESP, MOODS Team, Inserm, Faculté de Pharmacie, Université Paris-Saclay, Châtenay-Malabry, France.

Corinne Huchet (C)

TaRGeT, INSERM UMR 1089, Nantes Université, CHU Nantes, Nantes, France.

Aude Lafoux (A)

Therassay Platform, Capacités, Université de Nantes, IRS 2 Nantes Biotech, Nantes, France.

Rémi Mounier (R)

Institut NeuroMyoGène, Unité Physiopathologie et Génétique du Neurone et du Muscle, Université Claude Bernard Lyon 1, CNRS UMR 5261, Inserm U1315, Univ Lyon, Lyon, France.

Olivier Mir (O)

Sarcoma Group, Gustave Roussy, Villejuif, France.

Raphaël Gaillard (R)

GHU Paris Psychiatrie & Neurosciences, site Sainte Anne, Service Hospitalo-Universitaire de psychiatrie, Paris, France. raphael.gaillard@normalesup.org.
Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France. raphael.gaillard@normalesup.org.
Université de Paris Cité, Paris, France. raphael.gaillard@normalesup.org.

Fabrice Chrétien (F)

Institut Pasteur, Experimental Neuropathology Unit, Global Health Department, Paris, France. f.chretien@ghu-paris.fr.
Université de Paris Cité, Paris, France. f.chretien@ghu-paris.fr.
GHU Paris Psychiatrie & Neurosciences, site Sainte Anne, Service Hospitalo-Universitaire de neuropathologie, Paris, France. f.chretien@ghu-paris.fr.

Articles similaires

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male
Humans Meals Time Factors Female Adult

Classifications MeSH