The CD47/TSP-1 axis: a promising avenue for ovarian cancer treatment and biomarker research.


Journal

Molecular cancer
ISSN: 1476-4598
Titre abrégé: Mol Cancer
Pays: England
ID NLM: 101147698

Informations de publication

Date de publication:
14 Aug 2024
Historique:
received: 13 05 2024
accepted: 25 07 2024
medline: 14 8 2024
pubmed: 14 8 2024
entrez: 13 8 2024
Statut: epublish

Résumé

Ovarian cancer (OC) remains one of the most challenging and deadly malignancies facing women today. While PARP inhibitors (PARPis) have transformed the treatment landscape for women with advanced OC, many patients will relapse and the PARPi-resistant setting is an area of unmet medical need. Traditional immunotherapies targeting PD-1/PD-L1 have failed to show any benefit in OC. The CD47/TSP-1 axis may be relevant in OC. We aimed to describe changes in CD47 expression with platinum therapy and their relationship with immune features and prognosis. Tumor and blood samples collected from OC patients in the CHIVA trial were assessed for CD47 and TSP-1 before and after neoadjuvant chemotherapy (NACT) and multiplex analysis was used to investigate immune markers. Considering the therapeutic relevance of targeting the CD47/TSP-1 axis, we used the CD47-derived TAX2 peptide to selectively antagonize it in a preclinical model of aggressive ovarian carcinoma. Significant reductions in CD47 expression were observed post NACT. Tumor patients having the highest CD47 expression profile at baseline showed the greatest CD4 Our study thus (1) proposes a CD47-based stratification of patients who may be most likely to benefit from postoperative immunotherapy, and (2) suggests that TAX2 is a potential alternative therapy for patients relapsing on PARP inhibitors.

Sections du résumé

BACKGROUND BACKGROUND
Ovarian cancer (OC) remains one of the most challenging and deadly malignancies facing women today. While PARP inhibitors (PARPis) have transformed the treatment landscape for women with advanced OC, many patients will relapse and the PARPi-resistant setting is an area of unmet medical need. Traditional immunotherapies targeting PD-1/PD-L1 have failed to show any benefit in OC. The CD47/TSP-1 axis may be relevant in OC. We aimed to describe changes in CD47 expression with platinum therapy and their relationship with immune features and prognosis.
METHODS METHODS
Tumor and blood samples collected from OC patients in the CHIVA trial were assessed for CD47 and TSP-1 before and after neoadjuvant chemotherapy (NACT) and multiplex analysis was used to investigate immune markers. Considering the therapeutic relevance of targeting the CD47/TSP-1 axis, we used the CD47-derived TAX2 peptide to selectively antagonize it in a preclinical model of aggressive ovarian carcinoma.
RESULTS RESULTS
Significant reductions in CD47 expression were observed post NACT. Tumor patients having the highest CD47 expression profile at baseline showed the greatest CD4
CONCLUSIONS CONCLUSIONS
Our study thus (1) proposes a CD47-based stratification of patients who may be most likely to benefit from postoperative immunotherapy, and (2) suggests that TAX2 is a potential alternative therapy for patients relapsing on PARP inhibitors.

Identifiants

pubmed: 39138571
doi: 10.1186/s12943-024-02073-0
pii: 10.1186/s12943-024-02073-0
doi:

Substances chimiques

CD47 Antigen 0
Biomarkers, Tumor 0
CD47 protein, human 0
Thrombospondin 1 0
thrombospondin-1, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

166

Informations de copyright

© 2024. The Author(s).

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Auteurs

Aurélie Moniot (A)

Apmonia Therapeutics, Reims, France.

Christophe Schneider (C)

UMR 7369 MEDyC, CNRS, Université de Reims Champagne-Ardenne, Reims, France.

Laure Chardin (L)

Gustave-Roussy Cancer Campus Université Paris-Saclay GINECO/GINEGEPS, Inserm U981, Villejuif, France.

Elisa Yaniz-Galende (E)

Gustave-Roussy Cancer Campus Université Paris-Saclay GINECO/GINEGEPS, Inserm U981, Villejuif, France.

Catherine Genestie (C)

Gustave-Roussy Cancer Campus Université Paris-Saclay GINECO/GINEGEPS, Inserm U981, Villejuif, France.

Marion Etiennot (M)

Apmonia Therapeutics, Reims, France.

Aubéri Henry (A)

Apmonia Therapeutics, Reims, France.

Coralie Drelon (C)

UMR 7369 MEDyC, CNRS, Université de Reims Champagne-Ardenne, Reims, France.

Audrey Le Formal (A)

Gustave-Roussy Cancer Campus Université Paris-Saclay GINECO/GINEGEPS, Inserm U981, Villejuif, France.

Benoit Langlois (B)

UMR 7369 MEDyC, CNRS, Université de Reims Champagne-Ardenne, Reims, France.

Laurence Venat (L)

Centre Hospitalier Universitaire Dupuytren, Limoges, France.

Christophe Louvet (C)

Institut Mutualiste Montsouris-Jourdan, Paris, France.

Laure Favier (L)

Centre Georges-François Leclerc, Dijon, France.

Alain Lortholary (A)

Hôpital Privé du Confluent - GINECO, Nantes, France.

Dominique Berton-Rigaud (D)

ICO Centre René Gauducheau - GINECO, Saint-Herblain, France.

Nadine Dohollou (N)

Polyclinique Bordeaux Nord, Bordeaux, France.

Christophe Desauw (C)

Centre Hospitalier Régional Universitaire de Lille, Hôpital Huriez, Lille, France.

Michel Fabbro (M)

ICM Val d'Aurelle - GINECO, Montpellier, France.

Emmanuelle Malaurie (E)

Centre Hospitalier Intercommunal de Créteil, Créteil, France.

Coraline Dubot (C)

Institut Curie - Hôpital René Huguenin - GINECO, Saint-Cloud, France.

Jean Emmanuel Kurtz (JE)

Hôpitaux Universitaires de Strasbourg, Strasbourg, France.

Nathalie Bonichon Lamichhane (N)

Clinique Tivoli-Ducos, Bordeaux, France.

Éric Pujade-Lauraine (É)

ARCAGY-GINECO, Paris, France.

Albin Jeanne (A)

Apmonia Therapeutics, Reims, France.

Alexandra Leary (A)

Gustave-Roussy Cancer Campus Université Paris-Saclay GINECO/GINEGEPS, Inserm U981, Villejuif, France.

Stéphane Dedieu (S)

UMR 7369 MEDyC, CNRS, Université de Reims Champagne-Ardenne, Reims, France. stephane.dedieu@univ-reims.fr.

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