Certaines complications peuvent être gérées, mais d'autres peuvent être permanentes.
RéversibilitéGestion des complications
Facteurs de risque
5
#1
Quels sont les facteurs de risque pour les anomalies des motifs EF Hands ?
Les facteurs incluent des prédispositions génétiques et des expositions environnementales.
Facteurs de risquePrédispositions génétiques
#2
L'âge influence-t-il les motifs EF Hands ?
Oui, le vieillissement peut affecter la fonction des motifs EF Hands et la signalisation calcique.
ÂgeVieillissement
#3
Le mode de vie joue-t-il un rôle dans les risques ?
Un mode de vie sédentaire et une mauvaise alimentation augmentent les risques.
Mode de vieAlimentation
#4
Les antécédents familiaux sont-ils un facteur de risque ?
Oui, des antécédents familiaux de maladies génétiques peuvent augmenter le risque.
Antécédents familiauxMaladies génétiques
#5
Les infections peuvent-elles affecter les motifs EF Hands ?
Certaines infections peuvent perturber la signalisation calcique, affectant ces motifs.
InfectionsSignalisation calcique
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"position": 6,
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"name": "Les motifs EF Hands sont-ils liés à des maladies ?",
"position": 7,
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"@type": "Question",
"name": "Comment les motifs EF Hands affectent-ils la signalisation cellulaire ?",
"position": 8,
"acceptedAnswer": {
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"name": "Y a-t-il des signes cliniques de dysfonction des motifs EF Hands ?",
"position": 9,
"acceptedAnswer": {
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"@type": "Question",
"name": "Les motifs EF Hands influencent-ils le métabolisme ?",
"position": 10,
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"name": "Peut-on prévenir les anomalies des motifs EF Hands ?",
"position": 11,
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"name": "Y a-t-il des facteurs environnementaux à considérer ?",
"position": 12,
"acceptedAnswer": {
"@type": "Answer",
"text": "Oui, l'exposition à des toxines peut affecter la fonction des motifs EF Hands."
}
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"@type": "Question",
"name": "Comment le stress influence-t-il les motifs EF Hands ?",
"position": 13,
"acceptedAnswer": {
"@type": "Answer",
"text": "Le stress peut perturber la signalisation calcique, affectant indirectement ces motifs."
}
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"@type": "Question",
"name": "L'exercice physique a-t-il un impact sur ces motifs ?",
"position": 14,
"acceptedAnswer": {
"@type": "Answer",
"text": "L'exercice régule la signalisation calcique, ce qui peut soutenir la fonction des motifs."
}
},
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"@type": "Question",
"name": "Les suppléments de calcium sont-ils bénéfiques ?",
"position": 15,
"acceptedAnswer": {
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"text": "Des suppléments peuvent aider à maintenir un bon équilibre calcique, soutenant la fonction."
}
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"@type": "Question",
"name": "Quels traitements ciblent les motifs EF Hands ?",
"position": 16,
"acceptedAnswer": {
"@type": "Answer",
"text": "Actuellement, il n'existe pas de traitements spécifiques pour les motifs EF Hands."
}
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{
"@type": "Question",
"name": "Peut-on modifier les motifs EF Hands par thérapie génique ?",
"position": 17,
"acceptedAnswer": {
"@type": "Answer",
"text": "La thérapie génique pourrait potentiellement corriger des mutations affectant ces motifs."
}
},
{
"@type": "Question",
"name": "Les médicaments peuvent-ils influencer les motifs EF Hands ?",
"position": 18,
"acceptedAnswer": {
"@type": "Answer",
"text": "Certains médicaments modulent la signalisation calcique, indirectement affectant ces motifs."
}
},
{
"@type": "Question",
"name": "Y a-t-il des approches expérimentales pour traiter les anomalies ?",
"position": 19,
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"@type": "Answer",
"text": "Des recherches sont en cours sur des peptides et des inhibiteurs ciblant les motifs EF Hands."
}
},
{
"@type": "Question",
"name": "Comment la nutrition peut-elle influencer les motifs EF Hands ?",
"position": 20,
"acceptedAnswer": {
"@type": "Answer",
"text": "Une nutrition adéquate en calcium et en magnésium peut soutenir leur fonction."
}
},
{
"@type": "Question",
"name": "Quelles complications peuvent survenir avec des motifs EF Hands défectueux ?",
"position": 21,
"acceptedAnswer": {
"@type": "Answer",
"text": "Des complications peuvent inclure des troubles neurologiques et musculaires."
}
},
{
"@type": "Question",
"name": "Les motifs EF Hands sont-ils liés à des cancers ?",
"position": 22,
"acceptedAnswer": {
"@type": "Answer",
"text": "Des études suggèrent un lien potentiel entre des anomalies et certains types de cancer."
}
},
{
"@type": "Question",
"name": "Comment les complications affectent-elles la qualité de vie ?",
"position": 23,
"acceptedAnswer": {
"@type": "Answer",
"text": "Elles peuvent entraîner des limitations fonctionnelles et des douleurs chroniques."
}
},
{
"@type": "Question",
"name": "Y a-t-il des risques de maladies cardiovasculaires ?",
"position": 24,
"acceptedAnswer": {
"@type": "Answer",
"text": "Des dysfonctionnements calciques peuvent contribuer à des maladies cardiovasculaires."
}
},
{
"@type": "Question",
"name": "Les complications sont-elles réversibles ?",
"position": 25,
"acceptedAnswer": {
"@type": "Answer",
"text": "Certaines complications peuvent être gérées, mais d'autres peuvent être permanentes."
}
},
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"name": "Quels sont les facteurs de risque pour les anomalies des motifs EF Hands ?",
"position": 26,
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"text": "Les facteurs incluent des prédispositions génétiques et des expositions environnementales."
}
},
{
"@type": "Question",
"name": "L'âge influence-t-il les motifs EF Hands ?",
"position": 27,
"acceptedAnswer": {
"@type": "Answer",
"text": "Oui, le vieillissement peut affecter la fonction des motifs EF Hands et la signalisation calcique."
}
},
{
"@type": "Question",
"name": "Le mode de vie joue-t-il un rôle dans les risques ?",
"position": 28,
"acceptedAnswer": {
"@type": "Answer",
"text": "Un mode de vie sédentaire et une mauvaise alimentation augmentent les risques."
}
},
{
"@type": "Question",
"name": "Les antécédents familiaux sont-ils un facteur de risque ?",
"position": 29,
"acceptedAnswer": {
"@type": "Answer",
"text": "Oui, des antécédents familiaux de maladies génétiques peuvent augmenter le risque."
}
},
{
"@type": "Question",
"name": "Les infections peuvent-elles affecter les motifs EF Hands ?",
"position": 30,
"acceptedAnswer": {
"@type": "Answer",
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}
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RG Neuroplasticity, Leibniz Institute for Neurobiology, Magdeburg, Germany.
Leibniz Group 'Dendritic Organelles and Synaptic Function', University Medical Center Hamburg-Eppendorf, Center for Molecular Neurobiology, ZMNH, Hamburg, Germany.
Institute for Biological Instrumentation of the Russian Academy of Sciences, Federal Research Center Pushchino Scientific Center for Biological Research of the Russian Academy of Sciences, 142290 Pushchino, Russia. epermyak@yandex.ru.
Department of Biomedical Engineering, Pushchino State Institute of Natural Sciences, 142290 Pushchino, Russia. epermyak@yandex.ru.
The disease pathology for diabetes mellitus patients with chronic kidney disease (CKD) may be diabetic nephropathy (DN), non-diabetic renal disease (NDRD), or DN combined with NDRD. Considering that t...
We retrospectively screened 1,030 patients with type 2 diabetes who has undergone kidney biopsy from January 2005 to March 2017 in a single center. Variables were ranked according to importance, and t...
In total, 929 patients were assigned. Ten variables were selected for model development. The areas under the receiver operating characteristic curves (AUCROCs) for the RF and SVM methods were 0.953 an...
We successfully constructed a predictive model for DN and NDRD using machine learning methods, which were better than our regression methods....
ClinicalTrial.gov, NCT03865914....
To explore the characteristics of retina microvascular changes in patients with diabetic nephropathy (DN) and its risk factors....
Retrospective, observational study. 145 patients with type 2 diabetic mellitus (DM) and DN were included in the study. Demographic and clinical parameters were obtained from medical records. Presence ...
DR accounted for 61.4% in type 2 DM patients with DN, of which proliferative diabetic retinopathy (PDR) accounted for 23.6% and sight threatening DR accounted for 35.7%. DR group had significantly hig...
A relatively higher prevalence of DR was found in type 2 DM patients with DN. ACR stage could be recognized as a risk factor for DR in DN patients. Patients with DN needs ophthalmic examination more t...
Musclin is a recently found myokine involved in the process of glucose metabolism. The purpose of the present investigation is to evaluate the relationship between serum musclin levels and diabetic ne...
The current investigation included 175 (T2DM) cases and 62 controls. T2DM patients were divided into three subgroups: normoalbuminuria (DN0), microalbuminuria (DN1), and macroalbuminuria (DN2) on the ...
T2DM group displayed higher serum musclin than the controls. Serum musclin were remarkably elevated in DN2 subgroup compared with DN0 and DN1 subgroups. In addition, elevated serum musclin was observe...
Serum musclin increases with the progressed stages of DN. Serum musclin is associated with renal function parameters and ACR....
Diabetic nephropathy remains a significant economic and social burden on both the individual patient and health-care systems as the prevalence of diabetes increases in the general population. The comp...
We retrieved PubMed, Cochrane Library, Scopus, Google Scholar, and ClinicalTrials.gov records to identify studies and articles focused on new pharmacologic advances to treat diabetic nephropathy....
RAAS blockers have remained the mainstay of therapy for DM nephropathy for many years, with only recent advancements with SGLT2 inhibitors and nonsteroidal MRAs. Better understanding of the long-term ...
Diabetic nephropathy (DN) is one of the common chronic complications of diabetes with unclear molecular mechanisms, which is associated with end-stage renal disease (ESRD) and chronic kidney disease (...
Diabetic nephropathy is one of the most common and severe complications of diabetes mellitus, affecting one in every five patients suffering from diabetes. Despite extensive research, the exact pathog...
Diabetic nephropathy (DN) is the most common microvascular complication in diabetes and one of the leading causes of end-stage renal disease. The standard treatments for patients with classic DN focus...
This study aimed to analyze the distribution of IgG subclass in diabetic nephropathy (DN) and its association with clinicopathological features....
This is a single-center retrospective study enrolling 108 patients with biopsy-proven DN. Immunofluorescence and immunohistochemistry staining were applied, and clinicopathological features and renal ...
Both IgG and its subclasses colocalized with collagen IV α5 on glomerular basement membrane (GBM) and some of tubular basement membrane (TBM). IgG1 and the Mixed type were two predominant types of dep...
IgG subclass was selectively deposited along GBM and/or TBM in DN, and the mixed type of IgG subclass deposition on TBM had more clinical significance than the isotype and that on GBM. IgG subclass de...
Diabetic nephropathy (DKD) is the most common chronic microvascular complication of diabetes. About 20%-40% of diabetics develop DKD, which eventually leads to chronic kidney failure. Although progres...
The present study aimed to investigate and compare biomarkers of oxidative stress and the activity of antioxidant enzymes in the plasma of patients with different stages of diabetic nephropathy. For t...