Plasma Levels of the Bioactive Sphingolipid Metabolite S1P in Adult Cystic Fibrosis Patients: Potential Target for Immunonutrition?


Journal

Nutrients
ISSN: 2072-6643
Titre abrégé: Nutrients
Pays: Switzerland
ID NLM: 101521595

Informations de publication

Date de publication:
14 Mar 2020
Historique:
received: 17 01 2020
revised: 05 03 2020
accepted: 11 03 2020
entrez: 19 3 2020
pubmed: 19 3 2020
medline: 15 12 2020
Statut: epublish

Résumé

Recent research has linked sphingolipid (SL) metabolism with cystic fibrosis transmembrane conductance regulator (CFTR) activity, affecting bioactive lipid mediator sphingosine-1-phosphate (S1P). We hypothesize that loss of CFTR function in cystic fibrosis (CF) patients influenced plasma S1P levels. Total and unbound plasma S1P levels were measured in 20 lung-transplanted adult CF patients and 20 healthy controls by mass spectrometry and enzyme-linked immunosorbent assay (ELISA). S1P levels were correlated with CFTR genotype, routine laboratory parameters, lung function and pathogen colonization, and clinical symptoms. Compared to controls, CF patients showed lower unbound plasma S1P, whereas total S1P levels did not differ. A positive correlation of total and unbound S1P levels was found in healthy controls, but not in CF patients. Higher unbound S1P levels were measured in ΔF508-homozygous compared to ΔF508-heterozygous CF patients (

Identifiants

pubmed: 32183316
pii: nu12030765
doi: 10.3390/nu12030765
pmc: PMC7146441
pii:
doi:

Substances chimiques

CFTR protein, human 0
Lysophospholipids 0
Cystic Fibrosis Transmembrane Conductance Regulator 126880-72-6
sphingosine 1-phosphate 26993-30-6
Sphingosine NGZ37HRE42

Types de publication

Clinical Trial Comparative Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Austrian Science Fund
ID : KLI 284

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Auteurs

Emina Halilbasic (E)

Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, 1090 Vienna, Austria.

Elisabeth Fuerst (E)

Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, 1090 Vienna, Austria.

Denise Heiden (D)

Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, 1090 Vienna, Austria.

Lukasz Japtok (L)

Institute of Nutritional Science, Faculty of Mathematics and Natural Science, University of Potsdam, 14558 Nuthetal, Germany.

Susanne C Diesner (SC)

Department of Pediatrics and Adolescent Medicine, Medical University of Vienna, 1090 Vienna, Austria.

Michael Trauner (M)

Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, 1090 Vienna, Austria.

Askin Kulu (A)

Division of Thoracic Surgery, Department of Surgery, Medical University of Vienna, 1090 Vienna, Austria.

Peter Jaksch (P)

Division of Thoracic Surgery, Department of Surgery, Medical University of Vienna, 1090 Vienna, Austria.

Konrad Hoetzenecker (K)

Division of Thoracic Surgery, Department of Surgery, Medical University of Vienna, 1090 Vienna, Austria.

Burkhard Kleuser (B)

Institute of Nutritional Science, Faculty of Mathematics and Natural Science, University of Potsdam, 14558 Nuthetal, Germany.

Lili Kazemi-Shirazi (L)

Division of Gastroenterology and Hepatology, Department of Internal Medicine III, Medical University of Vienna, 1090 Vienna, Austria.

Eva Untersmayr (E)

Institute of Pathophysiology and Allergy Research, Center for Pathophysiology, Infectiology and Immunology, Medical University of Vienna, 1090 Vienna, Austria.

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Classifications MeSH