Rats with congenital hydronephrosis show increased susceptibility to renal ischemia-reperfusion injury.


Journal

Physiological reports
ISSN: 2051-817X
Titre abrégé: Physiol Rep
Pays: United States
ID NLM: 101607800

Informations de publication

Date de publication:
11 2020
Historique:
received: 18 05 2020
revised: 08 10 2020
accepted: 12 10 2020
entrez: 18 11 2020
pubmed: 19 11 2020
medline: 28 9 2021
Statut: ppublish

Résumé

Many drug candidates have shown significant renoprotective effects in preclinical models; however, there is no clinically used effective pharmacotherapy for acute kidney injury. The failure to translate from bench to bedside could be due to misleading results from experimental animals with undetected congenital kidney defects. This study was performed to assess the effects of congenital hydronephrosis on the functional capacity of tubular renal transporters as well as kidney sensitivity to ischemia-reperfusion (I-R)-induced injury in male Wistar rats. Ultrasonography was used to distinguish healthy control rats from rats with hydronephrosis. L-carnitine or furosemide was administered, and serial blood samples were collected and analyzed to assess the effects of hydronephrosis on the pharmacokinetic parameters. Renal injury was induced by clamping the renal pedicles of both kidneys for 30 min with subsequent 24 hr reperfusion. The prevalence of hydronephrosis reached ~30%. The plasma concentrations after administration of L-carnitine or furosemide were similar in both groups. I-R induced more pronounced renal injury in the hydronephrotic rats than the control rats, which was evident by a significantly higher kidney injury molecule-1 concentration and lower creatinine concentration in the urine of the hydronephrotic rats than the control rats. After I-R, the gene expression levels of renal injury markers were significantly higher in the hydronephrotic kidneys than in the kidneys of control group animals. In conclusion, our results demonstrate that hydronephrotic kidneys are more susceptible to I-R-induced damage than healthy kidneys. Unilateral hydronephrosis does not affect the pharmacokinetics of substances secreted or absorbed in the renal tubules.

Identifiants

pubmed: 33207081
doi: 10.14814/phy2.14638
pmc: PMC7673629
doi:

Substances chimiques

Cell Adhesion Molecules 0
Diuretics 0
Havcr1protein, rat 0
Furosemide 7LXU5N7ZO5
Carnitine S7UI8SM58A

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e14638

Informations de copyright

© 2020 The Authors. Physiological Reports published by Wiley Periodicals LLC on behalf of The Physiological Society and the American Physiological Society.

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Auteurs

Reinis Vilskersts (R)

Latvian Institute of Organic Synthesis, Riga, Latvia.
Rigas Stradins University, Riga, Latvia.

Karlis Vilks (K)

Latvian Institute of Organic Synthesis, Riga, Latvia.
University of Latvia, Riga, Latvia.

Melita Videja (M)

Latvian Institute of Organic Synthesis, Riga, Latvia.
Rigas Stradins University, Riga, Latvia.

Helena Cirule (H)

Latvian Institute of Organic Synthesis, Riga, Latvia.

Olga Zharkova-Malkova (O)

Latvian Institute of Organic Synthesis, Riga, Latvia.

Eduards Sevostjanovs (E)

Latvian Institute of Organic Synthesis, Riga, Latvia.

Maija Dambrova (M)

Latvian Institute of Organic Synthesis, Riga, Latvia.
Rigas Stradins University, Riga, Latvia.

Edgars Liepinsh (E)

Latvian Institute of Organic Synthesis, Riga, Latvia.

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Classifications MeSH