Fast and reliable Sanger POLE sequencing protocol in FFPE tissues of endometrial cancer.


Journal

Pathology, research and practice
ISSN: 1618-0631
Titre abrégé: Pathol Res Pract
Pays: Germany
ID NLM: 7806109

Informations de publication

Date de publication:
Feb 2023
Historique:
received: 12 11 2022
revised: 14 01 2023
accepted: 16 01 2023
pubmed: 5 2 2023
medline: 15 2 2023
entrez: 4 2 2023
Statut: ppublish

Résumé

The new classification of endometrial carcinoma (EC) requires molecular interpretation of somatic polymerase epsilon (POLE) exonuclease domain mutations. The identification of pathogenic mutations within the POLE gene defines the important subtype of ultramutated tumours ("POLE-ultramutated") with specified prognostic and predictive utility. POLE somatic mutations are present in 7-12% of ECs, usually high-grade tumours with aggressive appearance. Molecular analysis of the POLE gene can be performed using a qPCR test, the Sanger sequencing method, a next generation sequencing (NGS) panel test and also in situ hybridisation (IHC) assay. We describe our current approach of identification of POLE mutations using Sanger sequencing technology, which is still the most robust, accurate and fast technique to sequence DNA. We present a reliable protocol for Sanger sequencing of the entire sequence coding exonuclease domain of POLE - exons 9, 10, 11, 12, 13 and 14 (codons 268-491) with 5-10 nucleotides in exon/intron boundaries (reference sequences: NM_006231.4, NP_006222.2). The protocol has been optimized for formalin-fixed, paraffin-embedded (FFPE) EC tissues. The method developed in our laboratory allows better diagnosis of patients with EC according to current standards.

Sections du résumé

BACKGROUND BACKGROUND
The new classification of endometrial carcinoma (EC) requires molecular interpretation of somatic polymerase epsilon (POLE) exonuclease domain mutations. The identification of pathogenic mutations within the POLE gene defines the important subtype of ultramutated tumours ("POLE-ultramutated") with specified prognostic and predictive utility. POLE somatic mutations are present in 7-12% of ECs, usually high-grade tumours with aggressive appearance. Molecular analysis of the POLE gene can be performed using a qPCR test, the Sanger sequencing method, a next generation sequencing (NGS) panel test and also in situ hybridisation (IHC) assay. We describe our current approach of identification of POLE mutations using Sanger sequencing technology, which is still the most robust, accurate and fast technique to sequence DNA.
MATERIALS AND METHODS METHODS
We present a reliable protocol for Sanger sequencing of the entire sequence coding exonuclease domain of POLE - exons 9, 10, 11, 12, 13 and 14 (codons 268-491) with 5-10 nucleotides in exon/intron boundaries (reference sequences: NM_006231.4, NP_006222.2).
RESULT RESULTS
The protocol has been optimized for formalin-fixed, paraffin-embedded (FFPE) EC tissues.
CONCLUSION CONCLUSIONS
The method developed in our laboratory allows better diagnosis of patients with EC according to current standards.

Identifiants

pubmed: 36738508
pii: S0344-0338(23)00015-8
doi: 10.1016/j.prp.2023.154315
pii:
doi:

Substances chimiques

Poly-ADP-Ribose Binding Proteins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

154315

Informations de copyright

Copyright © 2023 Elsevier GmbH. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None.

Auteurs

Izabela Laczmanska (I)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland; Department of Genetics, Wroclaw Medical University, Marcinkowskiego 1, Wroclaw 50-368, Poland. Electronic address: laczmanska.izabela@dco.com.pl.

Dagmara Michalowska (D)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Marcin Jedryka (M)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland; Department of Oncology, Wroclaw Medical University, Hirszfelda 12, Wroclaw 53-413, Poland.

Dorota Blomka (D)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Mariola Semeniuk (M)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Ewelina Czykalko (E)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Mariola Abrahamowska (M)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Paulina Mlynarczykowska (P)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Agnieszka Chrusciel (A)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Ireneusz Pawlak (I)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland.

Adam Maciejczyk (A)

Lower Silesian Oncology, Pulmonology and Hematology Center, Hirszfelda 12, Wroclaw 53-413, Poland; Department of Oncology, Wroclaw Medical University, Hirszfelda 12, Wroclaw 53-413, Poland.

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