Titre : Mécanismes moléculaires de l'action pharmacologique

Mécanismes moléculaires de l'action pharmacologique : Questions médicales fréquentes

Termes MeSH sélectionnés :

Exercise Test
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"acceptedAnswer": { "@type": "Answer", "text": "L'imagerie par résonance magnétique et la tomographie permettent d'observer les effets des médicaments." } }, { "@type": "Question", "name": "Comment les biomarqueurs sont-ils utilisés dans le diagnostic ?", "position": 4, "acceptedAnswer": { "@type": "Answer", "text": "Les biomarqueurs aident à évaluer l'efficacité et la sécurité des médicaments en fonction de leur mécanisme." } }, { "@type": "Question", "name": "Quel rôle joue la pharmacogénomique dans le diagnostic ?", "position": 5, "acceptedAnswer": { "@type": "Answer", "text": "Elle permet d'adapter les traitements en fonction des variations génétiques influençant les mécanismes d'action." } }, { "@type": "Question", "name": "Quels symptômes indiquent un effet pharmacologique ?", "position": 6, "acceptedAnswer": { "@type": "Answer", "text": "Les symptômes peuvent inclure des changements physiologiques, des effets secondaires ou des améliorations cliniques." } }, { "@type": "Question", "name": "Comment les effets indésirables sont-ils liés aux mécanismes d'action ?", "position": 7, "acceptedAnswer": { "@type": "Answer", "text": "Les effets indésirables résultent souvent d'interactions non spécifiques avec des cibles moléculaires." } }, { "@type": "Question", "name": "Quels symptômes sont associés aux médicaments ciblant les récepteurs ?", "position": 8, "acceptedAnswer": { "@type": "Answer", "text": "Les symptômes peuvent inclure des modifications de la perception, de l'humeur ou de la douleur." } }, { "@type": "Question", "name": "Comment les symptômes varient-ils selon les mécanismes d'action ?", "position": 9, "acceptedAnswer": { "@type": "Answer", "text": "Les symptômes varient en fonction des cibles moléculaires et des voies de signalisation affectées." } }, { "@type": "Question", "name": "Quels symptômes peuvent indiquer une surdose ?", "position": 10, "acceptedAnswer": { "@type": "Answer", "text": "Des symptômes tels que des nausées, des vertiges ou des troubles de la conscience peuvent indiquer une surdose." } }, { "@type": "Question", "name": "Comment prévenir les effets indésirables des médicaments ?", "position": 11, "acceptedAnswer": { "@type": "Answer", "text": "Une évaluation des mécanismes d'action et des interactions médicamenteuses peut aider à prévenir les effets indésirables." } }, { "@type": "Question", "name": "Quel rôle joue l'éducation des patients dans la prévention ?", "position": 12, "acceptedAnswer": { "@type": "Answer", "text": "L'éducation des patients sur les mécanismes d'action des médicaments peut améliorer l'adhésion et réduire les risques." } }, { "@type": "Question", "name": "Comment les études cliniques aident-elles à prévenir les complications ?", "position": 13, "acceptedAnswer": { "@type": "Answer", "text": "Les études cliniques identifient les mécanismes d'action et les effets indésirables potentiels, permettant une meilleure prévention." } }, { "@type": "Question", "name": "Quels facteurs influencent la prévention des effets indésirables ?", "position": 14, "acceptedAnswer": { "@type": "Answer", "text": "Les facteurs incluent la génétique, l'âge, les comorbidités et les interactions médicamenteuses." } }, { "@type": "Question", "name": "Comment les recommandations de traitement aident-elles à prévenir les risques ?", "position": 15, "acceptedAnswer": { "@type": "Answer", "text": "Les recommandations basées sur les mécanismes d'action aident à choisir des traitements plus sûrs et efficaces." } }, { "@type": "Question", "name": "Comment les médicaments ciblent-ils des mécanismes spécifiques ?", "position": 16, "acceptedAnswer": { "@type": "Answer", "text": "Les médicaments sont conçus pour interagir avec des cibles moléculaires spécifiques, comme des enzymes ou des récepteurs." } }, { "@type": "Question", "name": "Quels traitements utilisent des mécanismes d'action innovants ?", "position": 17, "acceptedAnswer": { "@type": "Answer", "text": "Les thérapies géniques et les médicaments biologiques exploitent des mécanismes d'action novateurs." } }, { "@type": "Question", "name": "Comment les traitements sont-ils personnalisés ?", "position": 18, "acceptedAnswer": { "@type": "Answer", "text": "Les traitements sont adaptés en fonction des caractéristiques moléculaires des patients et des médicaments." } }, { "@type": "Question", "name": "Quel est l'impact des mécanismes d'action sur l'efficacité des traitements ?", "position": 19, "acceptedAnswer": { "@type": "Answer", "text": "Une compréhension approfondie des mécanismes d'action permet d'optimiser l'efficacité et de réduire les effets secondaires." } }, { "@type": "Question", "name": "Comment les combinaisons de médicaments exploitent les mécanismes d'action ?", "position": 20, "acceptedAnswer": { "@type": "Answer", "text": "Les combinaisons de médicaments peuvent cibler plusieurs mécanismes pour améliorer l'efficacité thérapeutique." } }, { "@type": "Question", "name": "Quelles complications peuvent survenir avec des médicaments ?", "position": 21, "acceptedAnswer": { "@type": "Answer", "text": "Les complications peuvent inclure des réactions allergiques, des effets toxiques ou des interactions médicamenteuses." } }, { "@type": "Question", "name": "Comment les mécanismes d'action influencent-ils les complications ?", "position": 22, "acceptedAnswer": { "@type": "Answer", "text": "Les mécanismes d'action peuvent déterminer la sévérité et la nature des complications observées." } }, { "@type": "Question", "name": "Quels sont les signes de complications graves ?", "position": 23, "acceptedAnswer": { "@type": "Answer", "text": "Des signes tels que des difficultés respiratoires, des éruptions cutanées ou des douleurs thoraciques peuvent indiquer des complications graves." } }, { "@type": "Question", "name": "Comment gérer les complications liées aux médicaments ?", "position": 24, "acceptedAnswer": { "@type": "Answer", "text": "La gestion des complications implique l'arrêt du médicament, le traitement des symptômes et la surveillance étroite." } }, { "@type": "Question", "name": "Quel rôle joue la pharmacovigilance dans la détection des complications ?", "position": 25, "acceptedAnswer": { "@type": "Answer", "text": "La pharmacovigilance surveille les effets indésirables et les complications pour améliorer la sécurité des médicaments." } }, { "@type": "Question", "name": "Quels facteurs de risque influencent l'efficacité des médicaments ?", "position": 26, "acceptedAnswer": { "@type": "Answer", "text": "Les facteurs incluent l'âge, le sexe, la génétique et les comorbidités qui peuvent affecter l'efficacité." } }, { "@type": "Question", "name": "Comment les interactions médicamenteuses sont-elles un facteur de risque ?", "position": 27, "acceptedAnswer": { "@type": "Answer", "text": "Les interactions médicamenteuses peuvent altérer les mécanismes d'action et augmenter le risque d'effets indésirables." } }, { "@type": "Question", "name": "Quel impact a l'alimentation sur les mécanismes d'action ?", "position": 28, "acceptedAnswer": { "@type": "Answer", "text": "L'alimentation peut influencer l'absorption et le métabolisme des médicaments, affectant leur efficacité." } }, { "@type": "Question", "name": "Comment le mode de vie affecte-t-il les traitements ?", "position": 29, "acceptedAnswer": { "@type": "Answer", "text": "Le mode de vie, y compris l'exercice et le tabagisme, peut modifier la réponse aux médicaments et leurs mécanismes d'action." } }, { "@type": "Question", "name": "Quels facteurs environnementaux influencent les effets des médicaments ?", "position": 30, "acceptedAnswer": { "@type": "Answer", "text": "Les facteurs environnementaux, comme la pollution, peuvent interagir avec les mécanismes d'action des médicaments." } } ] } ] }

Sous-catégories

86 au total
└─

Antagonistes des récepteurs aux angiotensines

Angiotensin Receptor Antagonists D057911 - D27.505.519.162
└─

Agents antiglycation

Antiglycation Agents D000093744 - D27.505.519.182
└─

Antagonistes des récepteurs de la bradykinine

Bradykinin Receptor Antagonists D065168 - D27.505.519.265
└─

Agents de déplétion en cystine

Cystine Depleting Agents D065104 - D27.505.519.334
└─

Inducteurs des enzymes du cytochrome P-450

Cytochrome P-450 Enzyme Inducers D065693 - D27.505.519.349
└─

Antagonistes des récepteurs de l'endothéline

Endothelin Receptor Antagonists D065128 - D27.505.519.364
└─

Activateurs d'enzymes

Enzyme Activators D020536 - D27.505.519.374
└─

Antienzymes

Enzyme Inhibitors D004791 - D27.505.519.389
└─

Réactivateurs d'enzymes

Enzyme Reactivators D004793 - D27.505.519.405
└─

Modulateurs de la fibrinolyse

Fibrin Modulating Agents D050299 - D27.505.519.421
└─

Modulateurs du transport transmembranaire

Membrane Transport Modulators D049990 - D27.505.519.562
└─

Modulateurs de la mitose

Mitosis Modulators D050258 - D27.505.519.593
└─

Peptidomimétiques

Peptidomimetics D057786 - D27.505.519.828
└─└─

Antagonistes du récepteur de type 1 de l'angiotensine-II

Angiotensin II Type 1 Receptor Blockers D047228 - D27.505.519.162.500
└─└─

Antagonistes du récepteur de type 2 de l'angiotensine-II

Angiotensin II Type 2 Receptor Blockers D057912 - D27.505.519.162.750
└─└─

Piégeurs de radicaux libres

Free Radical Scavengers D016166 - D27.505.519.217.500
└─└─

Antagonistes du récepteur B1 de la bradykinine

Bradykinin B1 Receptor Antagonists D065169 - D27.505.519.265.249
└─└─

Inducteurs du cytochrome P-450 CYP1A2

Cytochrome P-450 CYP1A2 Inducers D065694 - D27.505.519.349.120
└─└─

Inducteurs du cytochrome P-450 CYP2B6

Cytochrome P-450 CYP2B6 Inducers D065695 - D27.505.519.349.260
└─└─

Inducteurs du cytochrome P-450 CYP2C8

Cytochrome P-450 CYP2C8 Inducers D065696 - D27.505.519.349.308
└─└─

Inducteurs du cytochrome P-450 CYP2C9

Cytochrome P-450 CYP2C9 Inducers D065698 - D27.505.519.349.320
└─└─

Inducteurs du cytochrome P-450 CYP2C19

Cytochrome P-450 CYP2C19 Inducers D065697 - D27.505.519.349.350
└─└─

Inducteurs du cytochrome P-450 CYP2D6

Cytochrome P-450 CYP2D6 Inducers D065699 - D27.505.519.349.500
└─└─

Inducteurs du cytochrome P-450 CYP2E1

Cytochrome P-450 CYP2E1 Inducers D065700 - D27.505.519.349.600
└─└─

Inducteurs du cytochrome P-450 CYP3A

Cytochrome P-450 CYP3A Inducers D065701 - D27.505.519.349.700
└─└─

Antagonistes du récepteur de type A de l'endothéline

Endothelin A Receptor Antagonists D065130 - D27.505.519.364.500
└─└─

Antagonistes du récepteur de type B de l'endothéline

Endothelin B Receptor Antagonists D065131 - D27.505.519.364.750
└─└─

Activateurs de la Carbamoyl phosphate synthetase I

Carbamoyl Phosphate Synthetase I Activators D065097 - D27.505.519.374.099
└─└─

Activateurs de la GTPase

GTP Phosphohydrolase Activators D020549 - D27.505.519.374.200
└─└─

Inhibiteurs des protéines activatrices de la 5-lipoxygénase

5-Lipoxygenase-Activating Protein Inhibitors D058946 - D27.505.519.389.086
└─└─

Inhibiteurs de l'acétaldéhyde déshydrogénase

Acetaldehyde Dehydrogenase Inhibitors D065086 - D27.505.519.389.089
└─└─

Inhibiteurs de l'adénosine désaminase

Adenosine Deaminase Inhibitors D058892 - D27.505.519.389.092
└─└─

Inhibiteurs des adénylate cyclases

Adenylyl Cyclase Inhibitors D000067956 - D27.505.519.389.108
└─└─

Inhibiteurs de la calcineurine

Calcineurin Inhibitors D065095 - D27.505.519.389.174
└─└─

Inhibiteurs de l'anhydrase carbonique

Carbonic Anhydrase Inhibitors D002257 - D27.505.519.389.200
└─└─

Inhibiteurs et modulateurs de la gamma-secrétase

Gamma Secretase Inhibitors and Modulators D000091062 - D27.505.519.389.315
└─└─

Inhibiteurs des enzymes du cytochrome P-450

Cytochrome P-450 Enzyme Inhibitors D065607 - D27.505.519.389.335
└─└─

Antifoliques

Folic Acid Antagonists D005493 - D27.505.519.389.350
└─└─

Inhibiteurs de désacétylase d'histone

Histone Deacetylase Inhibitors D056572 - D27.505.519.389.360
└─└─

Inhibiteurs de l'intégrase

Integrase Inhibitors D019429 - D27.505.519.389.375
└─└─

Inhibiteurs de la lipoxygénase

Lipoxygenase Inhibitors D016859 - D27.505.519.389.480
└─└─

Inhibiteurs de la monoamine oxydase

Monoamine Oxidase Inhibitors D008996 - D27.505.519.389.616
└─└─

Inhibiteurs de la synthèse d'acide nucléique

Nucleic Acid Synthesis Inhibitors D019384 - D27.505.519.389.675
└─└─

Inhibiteurs de l'ornithine décarboxylase

Ornithine Decarboxylase Inhibitors D065108 - D27.505.519.389.705
└─└─

Inhibiteurs de la phosphodiestérase

Phosphodiesterase Inhibitors D010726 - D27.505.519.389.735
└─└─

Inhibiteurs des phosphoinositide-3 kinases

Phosphoinositide-3 Kinase Inhibitors D000081082 - D27.505.519.389.736
└─└─

Inhibiteurs de la phospholipase A2

Phospholipase A2 Inhibitors D064801 - D27.505.519.389.737
└─└─

Inhibiteurs de prolyle hydroxylases

Prolyl-Hydroxylase Inhibitors D064800 - D27.505.519.389.740
└─└─

Inhibiteurs de protéases

Protease Inhibitors D011480 - D27.505.519.389.745
└─└─

Inhibiteurs de protéines kinases

Protein Kinase Inhibitors D047428 - D27.505.519.389.755
└─└─

Inhibiteurs de la synthèse protéique

Protein Synthesis Inhibitors D011500 - D27.505.519.389.760
└─└─

Inhibiteurs de la pompe à protons

Proton Pump Inhibitors D054328 - D27.505.519.389.848
└─└─

Agents découplants

Uncoupling Agents D014475 - D27.505.519.389.936
└─└─

Agonistes de canaux chlorure

Chloride Channel Agonists D065101 - D27.505.519.562.311
└─└─

Agonistes de canaux sodiques

Sodium Channel Agonists D062687 - D27.505.519.562.625
└─└─

Mitogènes

Mitogens D008934 - D27.505.519.593.624
└─└─

Gazotransmetteurs

Gasotransmitters D064426 - D27.505.519.625.270
└─└─└─

Inhibiteurs de la synthèse d'acides gras

Fatty Acid Synthesis Inhibitors D054872 - D27.505.519.186.071.401
└─└─└─

Inhibiteurs du cytochrome P-450 CYP1A2

Cytochrome P-450 CYP1A2 Inhibitors D065609 - D27.505.519.389.335.120
└─└─└─

Inhibiteurs du cytochrome P-450 CYP2B6

Cytochrome P-450 CYP2B6 Inhibitors D065686 - D27.505.519.389.335.260
└─└─└─

Inhibiteurs du cytochrome P-450 CYP2C8

Cytochrome P-450 CYP2C8 Inhibitors D065687 - D27.505.519.389.335.308
└─└─└─

Inhibiteurs du cytochrome P-450 CYP2C19

Cytochrome P-450 CYP2C19 Inhibitors D065689 - D27.505.519.389.335.319
└─└─└─

Inhibiteurs du cytochrome P-450 CYP2C9

Cytochrome P-450 CYP2C9 Inhibitors D065688 - D27.505.519.389.335.329
└─└─└─

Inhibiteurs du cytochrome P-450 CYP2D6

Cytochrome P-450 CYP2D6 Inhibitors D065690 - D27.505.519.389.335.368
└─└─└─

Inhibiteurs du cytochrome P-450 CYP2E1

Cytochrome P-450 CYP2E1 Inhibitors D065691 - D27.505.519.389.335.421
└─└─└─

Inhibiteurs du cytochrome P-450 CYP3A

Cytochrome P-450 CYP3A Inhibitors D065692 - D27.505.519.389.335.503
└─└─└─

Inhibiteurs de la phosphodiestérase-3

Phosphodiesterase 3 Inhibitors D058987 - D27.505.519.389.735.249
└─└─└─

Inhibiteurs de la phosphodiestérase-4

Phosphodiesterase 4 Inhibitors D058988 - D27.505.519.389.735.374
└─└─└─

Inhibiteurs de la phosphodiestérase-5

Phosphodiesterase 5 Inhibitors D058986 - D27.505.519.389.735.500
└─└─└─

Inhibiteurs de l'enzyme de conversion de l'angiotensine

Angiotensin-Converting Enzyme Inhibitors D000806 - D27.505.519.389.745.085
└─└─└─

Inhibiteurs de la cystéine protéinase

Cysteine Proteinase Inhibitors D015853 - D27.505.519.389.745.325
└─└─└─

Inhibiteurs de métalloprotéinases matricielles

Matrix Metalloproteinase Inhibitors D061965 - D27.505.519.389.745.610
└─└─└─

Inhibiteurs du protéasome

Proteasome Inhibitors D061988 - D27.505.519.389.745.705
└─└─└─

Inhibiteurs de la rénine

Renin Inhibitors D000092502 - D27.505.519.389.745.753
└─└─└─

Inhibiteurs de la sérine protéinase

Serine Proteinase Inhibitors D015842 - D27.505.519.389.745.800
└─└─└─

Inhibiteurs des Janus kinases

Janus Kinase Inhibitors D000075242 - D27.505.519.389.755.500
└─└─└─

Inhibiteurs de protéine-tyrosine kinase

Tyrosine Protein Kinase Inhibitors D000092004 - D27.505.519.389.755.875
└─└─└─

Agonistes de canaux sodiques épithéliaux

Epithelial Sodium Channel Agonists D062708 - D27.505.519.562.625.249
└─└─└─

Agonistes de canaux sodiques voltage-dépendants

Voltage-Gated Sodium Channel Agonists D061585 - D27.505.519.562.625.500
└─└─└─

Inhibiteurs de canaux ioniques sensibles à l'acidité

Acid Sensing Ion Channel Blockers D062646 - D27.505.519.562.750.100
└─└─└─

Modulateurs de la polymérisation de la tubuline

Tubulin Modulators D050257 - D27.505.519.593.249.500
└─└─└─└─

Inhibiteurs des vasopeptidases

Vasopeptidase Inhibitors D000075563 - D27.505.519.389.745.085.500
└─└─└─└─

Inhibiteurs des caspases

Caspase Inhibitors D061945 - D27.505.519.389.745.325.500
└─└─└─└─

Inhibiteurs de serine peptidase de type Kazal

Serine Peptidase Inhibitors, Kazal Type D000074162 - D27.505.519.389.745.800.562
└─└─└─└─

Serpines

Serpins D015843 - D27.505.519.389.745.800.675
└─└─└─└─

Inhibiteurs trypsiques

Trypsin Inhibitors D014361 - D27.505.519.389.745.800.900

Sources (10000 au total)

Prognostic value of cardiopulmonary exercise test in children with congenital heart defects.

Cardiopulmonary exercise testing (CPET) has an important prognostic value in adults with different congenital heart defects (CHDs) and is a useful tool for risk stratification and clinical decision-ma... 411 CPET performed by paediatric patients with different CHDs were evaluated in this retrospective study. Medical records were reviewed to determine the presence of cardiac events. Participants were c... 411 patients with a median age at test of 12 years, 51 patients with simple CHD, 170 patients with moderate complexity CHD and 190 with high complexity CHD underwent CPET. Overall, CPET parameters wer... Reduced exercise capacity at young age is related to a higher probability of future cardiovascular events in paediatric patients with CHD. Submaximal exercise variables can be used instead when maxima...

Exercise testing to guide safe discharge from hospital in COVID-19: a scoping review to identify candidate tests.

We aimed to identify exercise tests that have been validated to support a safe discharge to home in patients with or without COVID-19.... Scoping review, using PRISMA-ScR reporting standards. Medline, PubMed, AMED, Embase, CINAHL and LitCovid databases were searched between 16 and 22 February 2021, with studies included from any publica... Short exercise tests.... Safe discharge from hospital, readmission rate, length of hospital stay, mortality. Secondary outcomes measures: safety, feasibility and reliability.... Of 1612 original records screened, 19 studies were included in the analysis. These used a variety of exercise tests in patients with chronic obstructive pulmonary disease, suspected pulmonary embolism... Further research is needed to aid clinical decision-making at discharge from hospital....

Cardiopulmonary exercise testing for patients with anorexia nervosa: a case-control study.

Patients with anorexia nervosa (AN) require appropriate nutrient therapy and physical activity management. Eating disorder treatment guidelines do not include safe, evidence-based intensity criteria f... CPX was performed with 14 female patients with AN admitted to a specialized eating disorder unit between 2015 and 2019. Their anaerobic threshold (AT) was determined by assessing their exercise tolera... The AT of the AN group (BMI: 15.7 [Mean] ± 1.8 standard deviation [SD]) was significantly lower than that of the HC group (BMI: 19.7 ± 1.8) (AN: 10.0 ± 1.8 vs. HC: 15.2 ± 3.0 ml/kg/min, P < 0.001). AT... This study showed that AT and AT-METS were lower in patients with AN than in HC. Patients with AN should be prescribed light-intensity aerobic exercise, and the current findings may help develop futur... III: Evidence obtained from case-control analytic studies....

Cardiopulmonary exercise testing in long covid shows the presence of dysautonomia or chronotropic incompetence independent of subjective exercise intolerance and fatigue.

After COVID-19 infection, 10-20% of patients suffer from varying symptoms lasting more than 12 weeks (Long COVID, LC). Exercise intolerance and fatigue are common in LC. The aim was to measure the max... Cardiopulmonary exercise test was conducted on 101 LC patients, who were admitted to exercise testing. The majority of them (86%) had been treated at home during their acute COVID-19 infection. Peak o... The measurements were performed in mean 12.7 months (SD 5.75) after COVID-19 infection in patients with exercise intolerance (group EI, 19 patients), fatigue (group F, 31 patients), their combination ... This study on LC patients with different symptoms showed that cardiopulmonary exercise capacity was in mean normal, with increased sympathetic activity in most patients. However, we identified subgrou...

Test-retest reliability of a 30-min fixed perceived effort cycling exercise.

Using exercise protocols at a fixed rating of perceived effort (RPE) is a useful method for exploring the psychophysical influences on exercise performance. However, studies that have employed this pr... Eight recreationally active cyclists completed two identical ramped incremental trials on a cycle ergometer to identify gas exchange threshold (GET). A linear regression model plotted RPE responses du... All performance and gas parameters showed excellent levels of test-retest reliability (ICCs = > .900) across both intensities. Performance, gas-related measures, and heart rate averaged over the entir... Recreationally active cyclists can reliably replicate fixed perceived effort exercise across multiple visits when RPE is aligned to physiological thresholds. Some evidence suggests that exercise at RP...

[Role of cardiopulmonary exercise test in early diagnosis of pulmonary hypertension in scleroderma patients].

Pulmonary arterial hypertension (PAH) is a severe, high mortality and progressive disease. Early diagnosis and treatment improves the prognosis. Patients with scleroderma disease presents high risk of... Prospective study of 52 consecutive patients diagnosed of scleroderma in our health area, during 2 years (2018 and 2019). All of them undergo CPET, in addition to the annual systematic screening. Sens... Fifty-two CPET were performed, of which 16 suggested PAH. Resting RHC confirmed PAH in 5 patients and exercise RHC in 7 (diagnostic sensitivity of CPET together with rest and exercise catheterization ... CPET and exercise RHC could detect PAH earlier than established screening in patients with scleroderma disease, allowing early diagnosis....

Cardiopulmonary exercise testing during follow-up after acute pulmonary embolism.

Cardiopulmonary exercise testing (CPET) may provide prognostically valuable information during follow-up after pulmonary embolism (PE). Our objective was to investigate the association of patterns and... In a prospective cohort study of unselected consecutive all-comers with PE, survivors of the index acute event underwent 3- and 12-month follow-ups, including CPET. We defined cardiopulmonary limitati... Overall, 396 patients were included. At 3 months, prevalence of cardiopulmonary limitation and deconditioning was 50.1% (34.7% mild/moderate; 15.4% severe) and 12.1%, respectively; at 12 months, it wa... Abnormal exercise capacity of cardiopulmonary origin is frequent after PE, being associated with clinical and haemodynamic impairment as well as long-term QoL reduction. CPET can be considered for sel...