EFTUD2 missense variants disrupt protein function and splicing in mandibulofacial dysostosis Guion-Almeida type.


Journal

Human mutation
ISSN: 1098-1004
Titre abrégé: Hum Mutat
Pays: United States
ID NLM: 9215429

Informations de publication

Date de publication:
08 2020
Historique:
received: 14 01 2020
revised: 26 03 2020
accepted: 19 04 2020
pubmed: 26 4 2020
medline: 9 11 2021
entrez: 26 4 2020
Statut: ppublish

Résumé

Pathogenic variants in the core spliceosome U5 small nuclear ribonucleoprotein gene EFTUD2/SNU114 cause the craniofacial disorder mandibulofacial dysostosis Guion-Almeida type (MFDGA). MFDGA-associated variants in EFTUD2 comprise large deletions encompassing EFTUD2, intragenic deletions and single nucleotide truncating or missense variants. These variants are predicted to result in haploinsufficiency by loss-of-function of the variant allele. While the contribution of deletions within EFTUD2 to allele loss-of-function are self-evident, the mechanisms by which missense variants are disease-causing have not been characterized functionally. Combining bioinformatics software prediction, yeast functional growth assays, and a minigene (MG) splicing assay, we have characterized how MFDGA missense variants result in EFTUD2 loss-of-function. Only four of 19 assessed missense variants cause EFTUD2 loss-of-function through altered protein function when modeled in yeast. Of the remaining 15 missense variants, five altered the normal splicing pattern of EFTUD2 pre-messenger RNA predominantly through exon skipping or cryptic splice site activation, leading to the introduction of a premature termination codon. Comparison of bioinformatic predictors for each missense variant revealed a disparity amongst different software packages and, in many cases, an inability to correctly predict changes in splicing subsequently determined by MG interrogation. This study highlights the need for laboratory-based validation of bioinformatic predictions for EFTUD2 missense variants.

Identifiants

pubmed: 32333448
doi: 10.1002/humu.24027
doi:

Substances chimiques

EFTUD2 protein, human 0
Peptide Elongation Factors 0
Ribonucleoprotein, U5 Small Nuclear 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1372-1382

Subventions

Organisme : Biotechnology and Biological Sciences Research Council
ID : BB/N000358/1
Pays : United Kingdom
Organisme : Department of Health
ID : IS‐BRC‐1215‐20007
Pays : United Kingdom

Informations de copyright

© 2020 The Authors. Human Mutation published by Wiley Periodicals, Inc.

Références

Bartels, C., Urlaub, H., Lührmann, R., & Fabrizio, P. (2003). Mutagenesis suggests several roles of Snu114p in pre-mRNA splicing. Journal of Biological Chemistry, 278(30), 28324-28334.
Beauchamp, M. C., Djedid, A., Daupin, K., Clokie, K., Kumar, S., Majewski, J., & Jerome-Majewska, L. A. (2019). Loss of function mutation of Eftud2, the gene responsible for mandibulofacial dysostosis with microcephaly (MFDM), leads to pre-implantation arrest in mouse. PLOS One, 14(7):e0219280. https://doi.org/10.1371/journal.pone.0219280
Brenner, T. J., & Guthrie, C. (2005). Genetic analysis reveals a role for the C terminus of the Saccharomyces cerevisiae GTPase Snu114 during spliceosome activation. Genetics, 170(3), 1063-1080.
Brummer, A., & Hausser, J. (2014). MicroRNA binding sites in the coding region of mRNAs: Extending the repertoire of post-transcriptional gene regulation. BioEssays, 36(6), 617-626. https://doi.org/10.1002/bies.201300104
Buratti, E., Chivers, M., Kralovicova, J., Romano, M., Baralle, M., Krainer, A. R., & Vorechovsky, I. (2007). Aberrant 5′ splice sites in human disease genes: Mutation pattern, nucleotide structure and comparison of computational tools that predict their utilization. Nucleic Acids Research, 35(13), 4250-4263. https://doi.org/10.1093/nar/gkm402
Cartegni, L., Chew, S. L., & Krainer, A. R. (2002). Listening to silence and understanding nonsense: Exonic mutations that affect splicing. Nature Reviews Genetics, 3(4), 285-298.
Deml, B., Reis, L. M., Muheisen, S., Bick, D., & Semina, E. V. (2015). EFTUD2 deficiency in vertebrates: Identification of a novel human mutation and generation of a zebrafish model. Birth defects research. Part A, Clinical and molecular teratology, 103(7), 630-640. https://doi.org/10.1002/bdra.23397
Desmet, F. O., Hamroun, D., Lalande, M., Collod-Beroud, G., Claustres, M., & Beroud, C. (2009). Human Splicing Finder: An online bioinformatics tool to predict splicing signals. Nucleic Acids Research, 37(9), e67. https://doi.org/10.1093/nar/gkp215
Fairbrother, W. G., Yeh, R. F., Sharp, P. A., & Burge, C. B. (2002). Predictive identification of exonic splicing enhancers in human genes. Science, 297(5583), 1007-1013. https://doi.org/10.1126/science.1073774
Frazer, L. N., Lovell, S. C., & O'Keefe, R. T. (2009). Analysis of synthetic lethality reveals genetic interactions between the GTPase Snu114p and snRNAs in the catalytic core of the Saccharomyces cerevisiae spliceosome. Genetics, 183, 497-515.
Frazer, L. N., Nancollis, V., & O'Keefe, R. T. (2008). The role of Snu114p during pre-mRNA splicing. Biochemical Society Transactions, 36(Pt 3), 551-553.
Gibson, D. G. (2011). Enzymatic assembly of overlapping DNA fragments. Method Enzymol, 498, 349-361. https://doi.org/10.1016/B978-0-12-385120-8.00015-2
Gordon, C. T., Petit, F., Oufadem, M., Decaestecker, C., Jourdain, A. S., Andrieux, J., … Amiel, J. (2012). EFTUD2 haploinsufficiency leads to syndromic oesophageal atresia. Journal of Medical Genetics, 49(12), 737-746. https://doi.org/10.1136/jmedgenet-2012-101173
Huang, L. J., Vanstone, M. R., Hartley, T., Osmond, M., Barrowman, N., Allanson, J., … Consortium, C. R. C. (2016). Mandibulofacial dysostosis with microcephaly: Mutation and database update. Human Mutation, 37(2), 148-154. https://doi.org/10.1002/humu.22924
Kishore, S., Khanna, A., & Stamm, S. (2008). Rapid generation of splicing reporters with pSpliceExpress. Gene, 427(1-2), 104-110. https://doi.org/10.1016/j.gene.2008.09.021
Kunkel, T. A. (1985). Rapid and efficient site-specific mutagenesis without phenotypic selection. Proceedings of the National Academy of Sciences of the United States of America, 82, 488-492.
Lacour, J. C., McBride, L., St Hilaire, H., Mundinger, G. S., Moses, M., Koon, J., … Lacassie, Y. (2019). Novel de novo EFTUD2 mutations in 2 cases with MFDM, initially suspected to have alternative craniofacial diagnoses. Cleft Palate-Craniofacial Journal, 56(5), 674-678. https://doi.org/10.1177/1055665618806379
Lehalle, D., Gordon, C. T., Oufadem, M., Goudefroye, G., Boutaud, L., Alessandri, J. L., … Amiel, J. (2014). Delineation of EFTUD2 haploinsufficiency-related phenotypes through a series of 36 patients. Human Mutation, 35(4), 478-485. https://doi.org/10.1002/humu.22517
Lehalle, D., Wieczorek, D., Zechi-Ceide, R. M., Passos-Bueno, M. R., Lyonnet, S., Amiel, J., & Gordon, C. T. (2015). A review of craniofacial disorders caused by spliceosomal defects. Clinical Genetics, 88(5), 405-415. https://doi.org/10.1111/cge.12596
Lei, L., Yan, S. Y., Yang, R., Chen, J. Y., Li, Y. M., Bu, Y., … Xiong, J. W. (2017). Spliceosomal protein eftud2 mutation leads to p53-dependent apoptosis in zebrafish neural progenitors. Nucleic Acids Research, 45(6), 3422-3436. https://doi.org/10.1093/nar/gkw1043
Lim, K. H., & Fairbrother, W. G. (2012). Spliceman-a computational web server that predicts sequence variations in pre-mRNA splicing. Bioinformatics, 28(7), 1031-1032. https://doi.org/10.1093/bioinformatics/bts074
Lines, M. A., Huang, L., Schwartzentruber, J., Douglas, S. L., Lynch, D. C., Beaulieu, C., … Boycott, K. M. (2012). Haploinsufficiency of a spliceosomal GTPase encoded by EFTUD2 causes mandibulofacial dysostosis with microcephaly. American Journal of Human Genetics, 90(2), 369-377. https://doi.org/10.1016/j.ajhg.2011.12.023
Luquetti, D. V., Hing, A. V., Rieder, M. J., Nickerson, D. A., Turner, E. H., Smith, J., … Cunningham, M. L. (2013). "Mandibulofacial dysostosis with microcephaly" caused by EFTUD2 mutations: Expanding the phenotype. American Journal of Medical Genetics, 161A(1), 108-113. https://doi.org/10.1002/ajmg.a.35696
Matsuo, M., Yamauchi, A., Ito, Y., Sakauchi, M., Yamamoto, T., Okamoto, N., … Saito, K. (2017). Mandibulofacial dysostosis with microcephaly: A case presenting with seizures. Brain and Development, 39(2), 177-181. https://doi.org/10.1016/j.braindev.2016.08.008
Mort, M., Sterne-Weiler, T., Li, B., Ball, E. V., Cooper, D. N., Radivojac, P., … Mooney, S. D. (2014). MutPred splice: Machine learning-based prediction of exonic variants that disrupt splicing. Genome Biology, 15(1), R19. https://doi.org/10.1186/gb-2014-15-1-r19
Nguyen, T. H. D., Galej, W. P., Bai, X. C., Oubridge, C., Newman, A. J., Scheres, S. H. W., & Nagai, K. (2016). Cryo-EM structure of the yeast U4/U6.U5 tri-snRNP at 3.7 angstrom resolution. Nature, 530(7590), 298-302. https://doi.org/10.1038/nature16940
Ni, W. J., & Leng, X. M. (2015). Dynamic miRNA-mRNA paradigms: New faces of miRNAs. Biochemistry and Biophysics Reports, 4, 337-341. https://doi.org/10.1016/j.bbrep.2015.10.011
Reese, M. G., Eeckman, F. H., Kulp, D., & Haussler, D. (1997). Improved splice site detection in Genie. Journal of Computational Biology, 4(3), 311-323. https://doi.org/10.1089/cmb.1997.4.311
Sarkar, A., Emrick, L. T., Smith, E. M., Austin, E. G., Yang, Y. P., Hunter, J. V., … Lalani, S. R. (2015). Novel de novo mutations in EFTUD2 detected by exome sequencing in mandibulofacial dysostosis with microcephaly syndrome. American Journal of Medical Genetics, 167(4), 914-918. https://doi.org/10.1002/ajmg.a.36948
Schwarz, J. M., Cooper, D. N., Schuelke, M., & Seelow, D. (2014). MutationTaster2: Mutation prediction for the deep-sequencing age. Nature Methods, 11(4), 361-362. https://doi.org/10.1038/nmeth.2890
Shapiro, M. B., & Senapathy, P. (1987). RNA splice junctions of different classes of eukaryotes: Sequence statistics and functional implications in gene expression. Nucleic Acids Research, 15(17), 7155-7174. https://doi.org/10.1093/nar/15.17.7155
Smigiel, R., Bezniakow, N., Jakubiak, A., Bloch, M., Patkowski, D., Obersztyn, E., & Sasiadek, M. M. (2015). Phenotype analysis of Polish patients with mandibulofacial dysostosis type Guion-Almeida associated with esophageal atresia and choanal atresia caused by EFTUD2 gene mutations. Journal of Applied Genetics, 56(2), 199-204. https://doi.org/10.1007/s13353-014-0255-4
Soukarieh, O., Gaildrat, P., Hamieh, M., Drouet, A., Baert-Desurmont, S., Frebourg, T., … Martins, A. (2016). Exonic splicing mutations are more prevalent than currently estimated and can be predicted by using in silico tools. PLOS Genetics, 12(1):e1005756. https://doi.org/10.1371/journal.pgen.1005756
Sterne-Weiler, T., & Sanford, J. R. (2014). Exon identity crisis: Disease-causing mutations that disrupt the splicing code. Genome Biology, 15(1), 1-8. https://doi.org/10.1186/Gb4150. Artn 201.
Vincent, M., Genevieve, D., Ostertag, A., Marlin, S., Lacombe, D., Martin-Coignard, D., … Collet, C. (2016). Treacher Collins syndrome: A clinical and molecular study based on a large series of patients. Genetics in Medicine, 18(1), 49-56. https://doi.org/10.1038/gim.2015.29
Voigt, C., Megarbane, A., Neveling, K., Czeschik, J. C., Albrecht, B., Callewaert, B., … Wieczorek, D. (2013). Oto-facial syndrome and esophageal atresia, intellectual disability and zygomatic anomalies-Expanding the phenotypes associated with EFTUD2 mutations. Orphanet Journal of Rare Diseases, 8, 110. https://doi.org/10.1186/1750-1172-8-110
Wahl, M. C., Will, C. L., & Lührmann, R. (2009). The spliceosome: Design principles of a dynamic RNP machine. Cell, 136(4), 701-718.
Will, C. L., & Lührmann, R. (2011). Spliceosome structure and function. Cold Spring Harbor Perspectives in Biology, 3(7), a003707. https://doi.org/10.1101/cshperspect.a003707
Wood, K. A., Rowlands, C. F., Qureshi, W. M. S., Thomas, H. B., Buczek, W. A., Briggs, T. A., … O'Keefe, R. T. (2019). Disease modelling of core pre-mRNA splicing factor haploinsufficiency. Human Molecular Genetics, 28, 3704-3723. https://doi.org/10.1093/hmg/ddz169
Xiong, H. Y., Alipanahi, B., Lee, L. J., Bretschneider, H., Merico, D., Yuen, R. K., … Frey, B. J. (2015). RNA splicing. The human splicing code reveals new insights into the genetic determinants of disease. Science, 347(6218):1254806. https://doi.org/10.1126/science.1254806
Yeo, G., & Burge, C. B. (2004). Maximum entropy modeling of short sequence motifs with applications to RNA splicing signals. Journal of Computational Biology, 11(2-3), 377-394. https://doi.org/10.1089/1066527041410418

Auteurs

Huw B Thomas (HB)

Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.

Katherine A Wood (KA)

Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.

Weronika A Buczek (WA)

Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.

Christopher T Gordon (CT)

Laboratory of Embryology and Genetics of Human Malformation, Institut National de la Santé et de la Recherche Médicale (INSERM) UMR 1163, Institut Imagine, Paris, France.
Paris Descartes-Sorbonne Paris Cité University, Institut Imagine, Paris, France.

Véronique Pingault (V)

Laboratory of Embryology and Genetics of Human Malformation, Institut National de la Santé et de la Recherche Médicale (INSERM) UMR 1163, Institut Imagine, Paris, France.
Paris Descartes-Sorbonne Paris Cité University, Institut Imagine, Paris, France.
Département de Génétique, Hôpital Necker-Enfants Malades, AP-HP, Paris, France.

Tania Attié-Bitach (T)

Paris Descartes-Sorbonne Paris Cité University, Institut Imagine, Paris, France.
Département de Génétique, Hôpital Necker-Enfants Malades, AP-HP, Paris, France.
INSERM UMR 1163, Institut Imagine, Paris, France.

Kathryn E Hentges (KE)

Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.

Vinod C Varghese (VC)

All Wales Medical Genomics Service, Cardiff, UK.

Jeanne Amiel (J)

Laboratory of Embryology and Genetics of Human Malformation, Institut National de la Santé et de la Recherche Médicale (INSERM) UMR 1163, Institut Imagine, Paris, France.
Paris Descartes-Sorbonne Paris Cité University, Institut Imagine, Paris, France.
Département de Génétique, Hôpital Necker-Enfants Malades, AP-HP, Paris, France.

William G Newman (WG)

Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.
Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, Center for Genomic Medicine, St. Mary's Hospital, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.

Raymond T O'Keefe (RT)

Division of Evolution and Genomic Sciences, School of Biological Sciences, Faculty of Biology, Medicine and Health, The University of Manchester, Manchester Academic Health Science Centre, Manchester, UK.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH